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Neurobiology of Disease|August 22, 2017
Cellular bases of the RNA metabolism dysfunction in motor neurons of a murine model of spinal muscular atrophy: Role of Cajal bodies and the nucleolusOlga Tapia, Josep Oriol Narcís, Javier Riancho, et al.Neurobiology of Disease|August 22, 2017
A deafness mechanism of digenic Cx26 (GJB2) and Cx30 (GJB6) mutations: Reduction of endocochlear potential by impairment of heterogeneous gap junctional function in the cochlear lateral wallLing Mei, Jin Chen, Liang Zong, et al.Neurobiology of Disease|August 24, 2017
Inhibition of tPA-induced hemorrhagic transformation involves adenosine A2b receptor activation after cerebral ischemiaQiang Li, Xiaoning Han, Xi Lan, et al.Neurobiology of Disease|August 29, 2017
Spatiotemporal resolution of spinal meningeal and parenchymal inflammation during experimental autoimmune encephalomyelitisBandana Shrestha, Xi Jiang, Shujun Ge, et al.Neurobiology of Disease|August 29, 2017
Protocadherin 10 alters γ oscillations, amino acid levels, and their coupling; baclofen partially restores these oscillatory deficitsRussell G Port, Christopher Gajewski, Elizabeth Krizman, et al.Neurobiology of Disease|October 7, 2017
Human amyloid β peptide and tau co-expression impairs behavior and causes specific gene expression changes in Caenorhabditis elegansChenyin Wang, Valeria Saar, Ka Lai Leung, et al.Neurobiology of Disease|September 1, 2017
Discovery of novel stroke-responsive lncRNAs in the mouse cortex using genome-wide RNA-seqSunil Bhattarai, Fabrizio Pontarelli, Emily Prendergast, et al.Neurobiology of Disease|September 2, 2017
Pro-excitatory alterations in sodium channel activity facilitate subiculum neuron hyperexcitability in temporal lobe epilepsyBryan S Barker, Aradhya Nigam, Matteo Ottolini, et al.Neurobiology of Disease|September 4, 2017
Differential deregulation of NGF and BDNF neurotrophins in a transgenic rat model of Alzheimer's diseaseM Florencia Iulita, M Beatriz Bistué Millón, Rowan Pentz, et al.Neurobiology of Disease|September 6, 2017
Inhibitory synapse deficits caused by familial α1 GABAA receptor mutations in epilepsyXiumin Chen, Nela Durisic, Joseph W Lynch, et al.Pageof 669