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The Journal of General Physiology|January 27, 2016
Orai1 pore residues control CRAC channel inactivation independently of calmodulinFranklin M Mullins, Michelle Yen, Richard S LewisThe Journal of General Physiology|January 27, 2016
The inactivation domain of STIM1 is functionally coupled with the Orai1 pore to enable Ca2+-dependent inactivationFranklin M Mullins, Richard S LewisThe Journal of General Physiology|January 13, 2016
Transition metal ion FRET to measure short-range distances at the intracellular surface of the plasma membraneSharona E Gordon, Eric N Senning, Teresa K Aman, et al.The Journal of General Physiology|February 17, 2016
Multiple cytosolic calcium buffers in posterior pituitary nerve terminalsShane M McMahon, Che-Wei Chang, Meyer B JacksonThe Journal of General Physiology|June 1, 1989
Tonic and phasic block of neuronal sodium currents by 5-hydroxyhexano-2',6'-xylide, a neutral lidocaine homologueD M Chernoff, G R StrichartzThe Journal of General Physiology|June 1, 1989
Calcium dependence of the activation and inactivation kinetics of the light-activated phosphodiesterase of retinal rodsA E Barkdoll, E N Pugh, A SitaramayyaThe Journal of General Physiology|October 28, 2015
Bimodal regulation of an Elk subfamily K+ channel by phosphatidylinositol 4,5-bisphosphateXiaofan Li, Andriy Anishkin, Hansi Liu, et al.The Journal of General Physiology|October 28, 2015
Modal affinities of endplate acetylcholine receptors caused by loop C mutationsRidhima Vij, Prasad Purohit, Anthony AuerbachThe Journal of General Physiology|September 30, 2015
Diffusion of myosin light chain kinase on actin: A mechanism to enhance myosin phosphorylation rates in smooth muscleFeng Hong, Richard K Brizendine, Michael S Carter, et al.The Journal of General Physiology|September 30, 2015
A novel Ca2+-feedback mechanism extends the operating range of mammalian rods to brighter lightFrans Vinberg, Teemu T Turunen, Hanna Heikkinen, et al.Pageof 668