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The Journal of Infectious Diseases|June 14, 2011
Stability of the age distribution of measles cases over time during outbreaks in Bangladesh, 2004-2006Eric Wiesen, Kathleen Wannemuehler, James L Goodson, et al.
The Journal of Infectious Diseases|June 14, 2011
Progress and challenges for measles elimination by 2012 in the Western Pacific RegionDavid H Sniadack, Jorge Mendoza-Aldana, Youngmee Jee, et al.
The Journal of Infectious Diseases|June 14, 2011
Progress toward measles elimination in the People's Republic of China, 2000-2009Chao Ma, Zhijie An, Lixin Hao, et al.
The Journal of Infectious Diseases|June 14, 2011
Epidemiology of a measles epidemic in Vietnam 2008-2010David H Sniadack, Jorge Mendoza-Aldana, Dang Thi Thanh Huyen, et al.
The Journal of Infectious Diseases|June 14, 2011
Two case studies of modified measles in vaccinated physicians exposed to primary measles cases: high risk of infection but low risk of transmissionJennifer S Rota, Carole J Hickman, Sun Bae Sowers, et al.
The Journal of Infectious Diseases|June 14, 2011
Dried blood spots on filter paper as an alternative specimen for measles diagnostics: detection of measles immunoglobulin M antibody by a commercial enzyme immunoassayAmra Uzicanin, Irene Lubega, Miriam Nanuynja, et al.
The Journal of Infectious Diseases|June 16, 2011
A modulatory interleukin-10 response to staphylococcal peptidoglycan prevents Th1/Th17 adaptive immunity to Staphylococcus aureusVanessa Frodermann, Thu A Chau, Samar Sayedyahossein, et al.
The Journal of Infectious Diseases|June 16, 2011
Role of a type IV-like secretion system of Streptococcus suis 2 in the development of streptococcal toxic shock syndromeYan Zhao, Gaoke Liu, Shu Li, et al.
The Journal of Infectious Diseases|June 16, 2011
Chemokine (C-C motif) receptor 5 -2459 genotype in patients receiving highly active antiretroviral therapy: race-specific influence on virologic successRajeev K Mehlotra, Vinay K Cheruvu, Melinda J Blood Zikursh, et al.
The Journal of Infectious Diseases|June 16, 2011
Endometrial epithelial cell responses to coinfecting viral and bacterial pathogens in the genital tract can activate the HIV-1 LTR in an NF{kappa}B-and AP-1-dependent mannerVictor H Ferreira, Aisha Nazli, Ghaznia Khan, et al.
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