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Megan M Simonds

6PUBLICATIONS
1CO-AUTHORS
Rheumatology and arthritisMedical infection agents (incl. prions)AutoimmunityLiquid biopsies
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Journal

Publications (6)

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|Mar 07, 2026
Soluble factors released from fibroblast-like synoviocytes of JIA patients with future extended course can condition persistent JIA synovial fibroblasts for increased IL-6 production.

Megan M Simonds, Lina Al-Jaberi, Hermine I Brunner

|Jan 03, 2024
Methotrexate inhibits BMP4 and abrogates the hypertrophic chondrocyte phenotype of synovial fibroblasts in juvenile idiopathic arthritis.

Megan M Simonds, Samuel T Freer, Anne Marie C Brescia

|Dec 09, 2023
Adalimumab Effectively Decreases Inflammation Downstream of TNFα Signaling in Synoviocytes from Extended Oligoarticular Juvenile Idiopathic Arthritis.

Megan M Simonds, Samuel T Freer, Lina Al-Jaberi

|Nov 07, 2022
CCL24, CXCL9 and CXCL10 are increased in synovial fluid in patients with juvenile idiopathic arthritis requiring advanced treatment.

Lina Al-Jaberi, Megan M Simonds, Anne Marie C Brescia

|May 13, 2021
The culture microenvironment of juvenile idiopathic arthritis synovial fibroblasts is favorable for endochondral bone formation through BMP4 and repressed by chondrocytes.

Megan M Simonds, Amanda R Schlefman, Suzanne M McCahan

|Nov 17, 2020
Juvenile idiopathic arthritis fibroblast-like synoviocytes influence chondrocytes to alter BMP antagonist expression demonstrating an interaction between the two prominent cell types involved in endochondral bone formation.

Megan M Simonds, Amanda R Schlefman, Suzanne M McCahan

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Frequent Collaborators

1 joint publications

Lina Al-Jaberi

Frequent Collaborators

1 joint publications

Lina Al-Jaberi

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