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[Selected lymphocyte subsets in Lime borreliosis: a preliminary study]

J M Zajkowska1, T Hermanowska-Szpakowicz, E Kasprzycka

  • 1Kliniki Chorób Pasozytniczych i Neuroinfekcji AM w Białymstoku. zajkowska@kki.net.pl

Insights

Lyme borreliosis significantly alters lymphocyte subsets, including CD4, CD8, and NK cells, before and after antibiotic treatment. These changes suggest a role in disease pathogenesis, with incomplete recovery indicating potential immune system inertness or insufficient therapy duration.

Area of Science:

  • Immunology
  • Infectious Diseases
  • Medical Research

Background:

  • Lyme borreliosis, caused by Borrelia burgdorferi, affects various bodily systems.
  • Immune system dysregulation, particularly in lymphocyte populations, is implicated in Lyme borreliosis pathogenesis.
  • Understanding changes in lymphocyte subsets can provide insights into disease progression and treatment efficacy.

Purpose of the Study:

  • To investigate the percentage changes in lymphocyte subsets (CD19, CD3, CD4, CD8, NK cells) and activated T cells (CD3+HLA-DR) in Lyme borreliosis patients.
  • To compare these immune parameters before and after a 4-week course of antibiotic therapy.
  • To evaluate the CD4/CD8 ratio and its changes in relation to treatment.

Main Methods:

  • Flow cytometric immunophenotyping was used to measure lymphocyte subsets and activated T cells.
  • 30 patients with confirmed Lyme borreliosis (erythema migrans, Lyme arthritis, neuroborreliosis) were studied.
  • Blood samples were analyzed before (I examination) and after (II examination) antibiotic treatment (ampicillin, ceftriaxone, or cefotaxime).

Main Results:

  • Significant decreases in CD19+ lymphocytes were observed after treatment compared to before.
  • Percentages of CD4, CD8, and NK cells were lower in patients compared to controls, both before and after treatment.
  • A lower CD4/CD8 ratio and reduced percentages of activated T cells (CD3+HLA-DR+) were noted post-treatment.
  • No significant changes were found in lymphocyte subsets with IL-2 receptors before and after treatment.

Conclusions:

  • Active Borrelia burgdorferi infection causes significant alterations in lymphocyte subsets.
  • Decreased percentages of CD4, CD8, NK cells, and activated T cells suggest their involvement in the immunopathogenesis of Lyme borreliosis.
  • The lack of full normalization of immune parameters post-treatment may be due to immune system inertness or inadequate antibiotic therapy duration.

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