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Effect of dietary ghee--the anhydrous milk fat on lymphocytes in rats

T G Niranjan1, T P Krishnakantha

  • 1Department of Biochemistry and Nutrition, Central Food Technological Research Institute, Mysore, India.

Insights

Dietary ghee consumption, particularly oxidized ghee, impairs lymphocyte function. This study shows ghee reduces lymphocyte proliferation and alters membrane structure, impacting immune response.

Area of Science:

  • Immunology
  • Nutrition Science
  • Biochemistry

Background:

  • Dietary lipids significantly influence immune system functioning.
  • Lymphocyte membrane lipid composition directly affects immune cell function.
  • Ghee, a clarified butter, is prevalent in Indian diets and contains saturated fatty acids and oxysterols.

Purpose of the Study:

  • To investigate the impact of fresh and oxidized ghee consumption on lymphocyte function and membrane composition in rats.
  • To determine if ghee alters lymphocyte proliferation, rosetting, and membrane integrity.

Main Methods:

  • Male weanling rats were fed diets containing fresh ghee, oxidized ghee, or groundnut oil (control) for 8 weeks.
  • Lymphocyte lipid composition, in vitro lipid peroxidation, Na+K+ ATPase activity, proliferation (using Con-A and PHA), and rosetting were analyzed.
  • Membrane fluidity was assessed in rats fed oxidized ghee.

Main Results:

  • Ghee consumption altered lymphocyte lipid composition.
  • Oxidized ghee significantly increased lymphocyte membrane lipid peroxidation (26%) and decreased Na+K+ ATPase activity (18%).
  • Lymphocyte proliferation decreased by 32% in ghee-fed rats, while rosetting decreased by 16% (fresh ghee) and 25% (oxidized ghee). Membrane fluidity declined in oxidized ghee-fed rats.

Conclusions:

  • Dietary ghee consumption, especially oxidized ghee, leads to decreased lymphocyte proliferation.
  • Oxysterols in oxidized ghee are implicated as inhibitors of lymphoproliferation.
  • Ghee consumption alters lymphocyte membrane structure and function, potentially compromising immune response.

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