Related Experiment Video

Updated: Jun 20, 2026

Detecting the Lyme Disease Spirochete, Borrelia Burgdorferi, in Ticks Using Nested PCR
07:20

Detecting the Lyme Disease Spirochete, Borrelia Burgdorferi, in Ticks Using Nested PCR

Published on: February 4, 2018

[Polymorphic mental disorders in the course of Lyme borreliosis--case study]

Bogusław Helon1, Tomasz Wojciech Tłuczek, Alina Buczyjan

  • 1Wojewódzki Podkarpacki Szpital Psychiatryczny w Zurawicy, Rózana.

Psychiatria Polska
|September 4, 2009
PubMed

Insights

This case study highlights how Lyme Borreliosis can manifest with varied psychiatric symptoms, including mood and paranoid disorders, before a physical diagnosis. Early recognition of these neurological signs is crucial for timely treatment.

Area of Science:

  • Neurology
  • Psychiatry
  • Infectious Diseases

Background:

  • Lyme Borreliosis is a tick-borne illness with diverse clinical presentations.
  • Neurological and psychiatric manifestations can precede or mimic primary mental health conditions.

Observation:

  • An inpatient case presented with initial qualitative consciousness disorders, followed by paranoid and depressive syndromes.
  • These psychiatric symptoms preceded the diagnosis of Lyme Borreliosis.

Findings:

  • The patient received therapy for Lyme Borreliosis.
  • Residual organic mood disorders were observed post-treatment, indicating lasting neurological impact.

Implications:

  • This case underscores the importance of considering Lyme Borreliosis in patients with unexplained polymorphic mental disorders.
  • Differential diagnosis in psychiatry should include infectious etiologies, particularly in endemic areas.
  • Timely diagnosis and treatment of Lyme Borreliosis can potentially mitigate severe or persistent psychiatric sequelae.

More Related Videos

Simultaneous Detection of Different Antibody Classes in a Multiplexed Serological Test05:25

Simultaneous Detection of Different Antibody Classes in a Multiplexed Serological Test

Published on: July 14, 2023

Feeding of Ticks on Animals for Transmission and Xenodiagnosis in Lyme Disease Research08:23

Feeding of Ticks on Animals for Transmission and Xenodiagnosis in Lyme Disease Research

Published on: August 31, 2013

Related Concept Videos

Bacterial Phylum Spirochaetes01:30

Bacterial Phylum Spirochaetes

Spirochetes, unique bacteria in the phylum Spirochaetes, are gram-negative, motile, tightly coiled, slender, and flexible. They inhabit aquatic sediments and animals, with some causing diseases like syphilis. Spirochetes are classified into eight genera based on habitat, pathogenicity, phylogeny, and characteristics.Their distinctive motility arises from endoflagella, located within the cell’s periplasm. These endoflagella anchor at the cell poles and extend along the cell length, encased by a...
Toxoplasmosis01:28

Toxoplasmosis

Toxoplasmosis, a zoonotic disease caused by the protozoan Toxoplasma gondii, poses significant public health challenges globally due to its high seroprevalence and varied clinical manifestations. As an obligate intracellular parasite, T. gondii can infect all warm-blooded vertebrates, but felids are its only definitive hosts, shedding unsporulated oocysts into the environment. Humans typically acquire the infection through ingestion of tissue cysts in undercooked meat or oocysts from...
Rocky Mountain Spotted Fever01:26

Rocky Mountain Spotted Fever

Rocky Mountain Spotted Fever (RMSF) is a severe tick-borne illness caused by Rickettsia rickettsii, a Gram-negative, coccobacillary bacterium. This pathogen is an obligate intracellular parasite, requiring a host cell for replication. Transmission occurs through the bite of an infected tick. In the United States, the most important vectors are Dermacentor variabilis (American dog tick) and Dermacentor andersoni (Rocky Mountain wood tick), though other tick species may also serve as vectors.
Encephalitis ll: Pathophysiology01:26

Encephalitis ll: Pathophysiology

Encephalitis is inflammation of the brain parenchyma caused by direct viral invasion or immune-mediated mechanisms triggered by infections or tumors. Both processes lead to neuronal injury, disrupted neurotransmission, and diverse neurological symptoms, often with overlapping clinical and pathological features.Autoimmune EncephalitisIn autoimmune encephalitis, antibodies target neuronal antigens on cell surfaces, synapses, or within neurons. A key example is anti-NMDAR encephalitis, which can...
Encephalitis l: Introduction01:19

Encephalitis l: Introduction

Encephalitis is inflammation of the brain parenchyma, most often due to infections or autoimmune processes. It presents with neuropsychiatric features such as fever, altered mental status, behavioral changes, cognitive dysfunction, seizures, focal deficits, and sometimes autonomic instability. In some cases, the meninges are also involved, resulting in meningoencephalitis.Infectious CausesInfectious encephalitis is most commonly viral but can also result from bacterial, fungal, or parasitic...
Bacterial Meningitis II: Pathophysiology01:26

Bacterial Meningitis II: Pathophysiology

Bacterial meningitis typically begins when pathogens such as Neisseria meningitidis and Streptococcus pneumoniae colonize the nasopharynx and invade the bloodstream. This process is facilitated by bacterial virulence factors, such as polysaccharide capsules, which resist phagocytosis and complement-mediated killing. Less commonly, bacteria reach the central nervous system via contiguous spread from infections like otitis media or sinusitis, through congenital or acquired dural defects, or...