Soluble CD16 binds peripheral blood mononuclear cells and inhibits pokeweed-mitogen-induced responses

C Teillaud1, J Galon, M T Zilber

  • 1Laboratoire d'Immunologie Cellulaire et Clinique, Unité INSERM 255, Institut Curie, Paris, France.

Blood
|November 15, 1993
PubMed

Insights

Soluble CD16 (sCD16), derived from neutrophils, binds to human immune cells and inhibits their proliferation and antibody production. This suggests neutrophils regulate immune responses through sCD16.

Area of Science:

  • Immunology
  • Cell Biology
  • Biochemistry

Background:

  • Neutrophils express Fc gamma RII (CD32) and Fc gamma RIIIB (CD16) low-affinity IgG receptors.
  • Soluble CD16 (sCD16) is generated from neutrophil Fc gamma RIIIB via proteolysis near the cell surface.

Purpose of the Study:

  • To produce recombinant soluble CD16 (sCD16) in eukaryotic cells.
  • To investigate the functional roles and binding properties of sCD16.
  • To assess the impact of sCD16 on human peripheral blood mononuclear cell (PBMC) responses.

Main Methods:

  • Production of recombinant sCD16 in eukaryotic cells.
  • Binding assays with human IgG subclasses and F(ab')2 fragments.
  • In vitro proliferation and antibody production assays using pokeweed mitogen (PWM) stimulated PBMCs.
  • Flow cytometry (FACS) analysis to determine sCD16 binding to PBMCs.

Main Results:

  • Purified recombinant sCD16 (48 kD) specifically bound human IgG1 and IgG3.
  • sCD16 inhibited PWM-induced PBMC proliferation and IgM/IgG production in a time- and dose-dependent manner.
  • FACS analysis confirmed sCD16 specifically binds to monocytes, B cells, and T cell subsets within PBMCs.

Conclusions:

  • Soluble CD16 (sCD16) exhibits cell-binding and antiproliferative capacities.
  • Neutrophils may regulate immune responses through the release of sCD16.
  • sCD16 exerts its biological functions through direct interactions with mononuclear blood cells.