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Lymphocyte traffic changes induced by monolateral vagal denervation in mouse thymus and peripheral lymphoid organs

A Antonica1, E Ayroldi, F Magni

  • 1Istituto di Patologia Generale, Cattedra de Fisiologia Umana, Universitá de Perugia, Italy.

Insights

Vagotomy (vagal nerve denervation) temporarily increases immature lymphocyte release from the thymus to the spleen and lymph nodes. This suggests the vagal nerve influences lymphocyte traffic within the thymus.

Area of Science:

  • Immunology
  • Neuroscience
  • Vascular Biology

Background:

  • The thymus is crucial for T-cell maturation and export.
  • The vagus nerve plays a role in regulating various physiological processes.

Purpose of the Study:

  • To investigate the effect of vagotomy on lymphocyte release from the mouse thymus.
  • To explore the role of the vagus nerve in immune cell trafficking.

Main Methods:

  • Cervical vagotomy was performed on mice.
  • Thymocytes were labeled in situ with fluorescein isothiocyanate (FITC).
  • Flow cytometry was used to analyze lymphocyte populations (CD4, CD8) in thymus, spleen, and lymph nodes.

Main Results:

  • Vagotomy transiently increased the export of immature CD4+CD8+ (DP) and CD4-CD8- (DN) thymocytes.
  • The percentage of mature CD4+CD8- and CD4-CD8+ (SP) thymocytes decreased in peripheral lymphoid organs.
  • Increased lymphocyte release was observed 24 and 48 hours post-vagotomy.

Conclusions:

  • The vagus nerve may facilitate the egress of mature lymphocytes from the thymus.
  • Vagotomy-induced changes in lymphocyte populations are likely not due to stress.
  • The vagal nerve's influence on lymphocyte traffic occurs at the thymic cortico-medullary junction.

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