Related Experiment Videos

ER-60, a chaperone with thiol-dependent reductase activity involved in MHC class I assembly

J A Lindquist1, O N Jensen, M Mann

  • 1Department of Molecular Immunology, German Cancer Research Center, In Neuenheimer Feld 280, 69120 Heidelberg, Germany.

The EMBO Journal
|May 26, 1998
PubMed

Insights

A new molecular chaperone, ER-60, is identified in the assembly of MHC class I molecules. ER-60, a thiol reductase, plays a role in heavy chain complex formation and association with TAP, even without beta2-microglobulin.

Area of Science:

  • Immunology
  • Molecular Biology
  • Protein Biochemistry

Background:

  • MHC class I assembly involves chaperones calnexin and calreticulin.
  • Association with the transporter associated with antigen processing (TAP) is crucial for antigen presentation.

Purpose of the Study:

  • To identify novel components in the MHC class I assembly pathway.
  • To elucidate the role of ER-60 in MHC class I complex formation and TAP association.

Main Methods:

  • Peptide mapping by matrix-assisted laser desorption/ionization (MALDI) mass spectrometry.
  • Analysis of MHC class I heavy chain complexes in a beta2-microglobulin-negative cell line.

Main Results:

  • ER-60 (thiol-dependent reductase) identified as a new molecular chaperone in MHC class I assembly.
  • ER-60 is found in early and late MHC class I assembly complexes with calnexin, calreticulin, tapasin, and TAP.
  • ER-60-calnexin-heavy chain complexes bind to TAP independently of beta2-microglobulin.

Conclusions:

  • ER-60 is a novel chaperone involved in MHC class I assembly and TAP association.
  • ER-60's thiol reductase activity may be critical for disulfide bond formation in heavy chains.
  • Beta2-microglobulin is not essential for the initial association of MHC class I heavy chains with TAP.

Related Concept Videos