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Lymphoproliferative disorders: CT findings in immunocompromised children

L F Donnelly1, D P Frush, K W Marshall

  • 1Department of Radiology, Duke University Medical Center, Durham, NC 27710, USA.

Insights

Epstein-Barr virus-induced lymphoproliferative disorders in children show varied CT imaging and distribution. Mortality is linked more to the type of immune dysfunction than disease spread, especially in transplant recipients.

Area of Science:

  • Pediatric Radiology
  • Immunology
  • Oncology

Background:

  • Epstein-Barr virus (EBV) can cause lymphoproliferative disorders (LPDs) in immunocompromised children.
  • Understanding the imaging features and prognostic factors of EBV-LPDs is crucial for timely diagnosis and management.

Purpose of the Study:

  • To evaluate the CT imaging characteristics, disease distribution, and outcomes of pediatric EBV-LPDs.
  • To correlate imaging findings and disease patterns with the type of immunocompromised state and patient prognosis.

Main Methods:

  • Retrospective review of medical records and CT imaging studies from four tertiary children's medical centers.
  • Analysis of pathologically proven EBV-LPD cases in patients under 20 years old.
  • Statistical analysis (Fisher's exact test) to identify trends between CT appearance, disease distribution, immune status, and prognosis.

Main Results:

  • Twenty-seven pediatric cases of EBV-LPD were identified, with a mean age of 7 years 8 months.
  • Common CT findings included lymphadenopathy, focal masses, and organ enlargement; abdominal involvement was most frequent.
  • Mortality rates (44%) were significantly associated with the type of immune dysfunction (e.g., 100% in bone marrow transplant recipients) rather than disease extent.

Conclusions:

  • Pediatric EBV-LPDs exhibit diverse CT appearances, distribution patterns, and outcomes.
  • In solid organ transplant recipients, EBV-LPDs often localized to the anatomical region of the transplant.
  • Prognosis is more strongly influenced by the underlying immune deficiency than the spatial distribution of the LPD.
Abstract

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