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Indometacina y el vaciado de la vesícula biliar postprandial.

L J O'Donnell1, P Wilson, P Guest

  • 1Department of Gastroenterology, St Bartholomew's Hospital, West Smithfield, London, UK.

Lancet (London, England)
|February 1, 1992
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Los antiinflamatorios no esteroideos (AINE) como la indometacina mejoran el vaciado de la vesícula biliar en pacientes con enfermedad de cálculos biliares. Esto sugiere que los AINE pueden prevenir la formación de cálculos biliares al mejorar la motilidad de la vesícula biliar.

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Área de la Ciencia:

  • Gastroenterología y Gastroenterología.
  • Farmacología Farmacología.

Sus antecedentes:

  • La enfermedad de cálculos biliares a menudo se asocia con una deficiencia en el vaciado de la vesícula biliar.
  • Los medicamentos antiinflamatorios no esteroideos (AINE) a veces están relacionados con la prevención de cálculos biliares, pero el mecanismo no está claro.

Objetivo del estudio:

  • Investigar si los AINE, específicamente la indometacina, pueden mejorar el vaciado de la vesícula biliar en pacientes con enfermedad de cálculos biliares.
  • Para determinar si este efecto difiere entre los pacientes con cálculos biliares y los individuos sanos.

Principales métodos:

  • Se realizó un estudio aleatorizado, controlado con placebo.
  • Los participantes recibieron indometacina (25 mg tres veces al día durante una semana) o un placebo durante una semana.
  • El vaciado de la vesícula biliar postprandial se evaluó en ambos grupos.

Principales resultados:

  • La indometacina mejoró significativamente el vaciado de la vesícula biliar postprandial en los 7 pacientes con enfermedad de cálculos biliares.
  • No se observó ningún efecto significativo en el vaciado de la vesícula biliar en sujetos sanos con función normal de la vesícula biliar.

Conclusiones:

  • Los AINE, como la indometacina, pueden prevenir la formación de cálculos biliares al actuar como un agente prokinético que mejora el vaciado de la vesícula biliar.
  • El efecto observado es específico para individuos con función de vesícula biliar deteriorada, lo que sugiere un mecanismo terapéutico dirigido.