Jove
Visualize
Contáctanos
JoVE
x logofacebook logolinkedin logoyoutube logo
ACERCA DE JoVE
Visión GeneralLiderazgoBlogCentro de Ayuda JoVE
AUTORES
Proceso de PublicaciónConsejo EditorialAlcance y PolíticasRevisión por ParesPreguntas FrecuentesEnviar
BIBLIOTECARIOS
TestimoniosSuscripcionesAccesoRecursosConsejo Asesor de BibliotecasPreguntas Frecuentes
INVESTIGACIÓN
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchivo
EDUCACIÓN
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualCentro de Recursos para ProfesoresSitio de Profesores
Términos y Condiciones de Uso
Política de Privacidad
Políticas

Videos de Conceptos Relacionados

Ischemic Stroke l: Introduction01:15

Ischemic Stroke l: Introduction

Ischemic stroke is an acute cerebrovascular condition in which blood flow to a brain region is suddenly interrupted, leading to tissue infarction. Neurons depend on continuous oxygen and glucose supply, so even brief reductions in perfusion cause energy failure, ionic imbalance, and irreversible injury. Ischemic strokes are classified into thrombotic and embolic types based on their underlying mechanisms.Thrombotic MechanismsThrombotic stroke develops when a clot forms within a cerebral artery.
Hemorrhagic Stroke l: Introduction01:17

Hemorrhagic Stroke l: Introduction

A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
Ischemic Stroke ll: Pathophysiology01:15

Ischemic Stroke ll: Pathophysiology

An ischemic stroke occurs when a cerebral blood vessel becomes obstructed, most often by a thrombus or embolus, interrupting the delivery of oxygen and glucose to brain tissue. Because neurons rely on continuous aerobic metabolism, energy failure begins within minutes of reduced perfusion. The region receiving the least blood flow becomes the infarct core, an area of irreversible cellular death. Surrounding this core lies the penumbra, a zone of hypoperfused but still viable tissue that is...
Venous Thrombosis III: Interprofessional Care01:29

Venous Thrombosis III: Interprofessional Care

Venous thrombosis requires effective prevention and treatment strategies to improve patient outcomes and reduce potential complications.Prevention StrategiesHealthcare providers must prioritize preventing venous thromboembolism (VTE) for all adult patients upon admission. Interventions depend on bleeding and thrombosis risk, medical history, current medications, diagnoses, planned procedures, and patient preferences. Patients on bed rest should change positions every two hours and, if not...
Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Venous Thrombosis IV: Nursing Management01:30

Venous Thrombosis IV: Nursing Management

Nursing management begins with a thorough assessment of the patient's health history. Key factors include trauma to veins, peripherally inserted central catheters, varicose veins, recent pregnancy or childbirth, surgery, bacteremia, prolonged bed rest, atrial fibrillation, COPD, heart failure, cancer, coagulation disorders, myocardial infarction, spinal cord injury, stroke, prolonged travel, recent bone fractures, and dehydration. Review medication intake, particularly oral contraceptives,...

También podría leer

Artículos Relacionados

Artículos vinculados a este trabajo por autores compartidos, revista y gráfico de citas.

Ordenar por
Same author

In-Stent Stenosis or Occlusion After Carotid Artery Stenting in Patients Treated With Anticoagulants vs Antiplatelet Therapy.

Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists·2026
Same author

Noninvasive neurophysiological diagnostics of delayed cerebral ischemia in aneurysmal subarachnoid hemorrhage: a scoping review.

Acta neurochirurgica·2026
Same author

Endovascular treatment in patients with renal dysfunction: results of the MR CLEAN Registry.

European stroke journal·2026
Same author

Cost-Effectiveness of Thrombectomy With or Without Alteplase in Large Vessel Occlusion Stroke: A Meta-Analysis Considering Time-to-Treatment.

Neurology·2026
Same author

Asymptomatic Carotid Stenosis and Cognition: A Contemporary Narrative Review of Cerebral Perfusion and Cerebrovascular Reserve.

Annals of vascular surgery·2026
Same author

Quality of life after decompressive surgery for severe cerebral venous thrombosis.

Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association·2026

Video Experimental Relacionado

Updated: May 18, 2026

A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia
06:01

A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia

Published on: August 18, 2015

Riesgo de sangrado con la terapia de accidente cerebrovascular isquémico.

Irene Miedema, Gert-Jan Luijckx, Maarten Uyttenboogaart

    JAMA
    |October 4, 2012
    PubMed
    Resumen

    No abstract available in PubMed .

    Más Videos Relacionados

    Real-Time Monitoring and Modulation of Blood Pressure in a Rabbit Model of Ischemic Stroke
    09:00

    Real-Time Monitoring and Modulation of Blood Pressure in a Rabbit Model of Ischemic Stroke

    Published on: February 10, 2023

    2-Vessel Occlusion/Hypotension: A Rat Model of Global Brain Ischemia
    09:29

    2-Vessel Occlusion/Hypotension: A Rat Model of Global Brain Ischemia

    Published on: June 22, 2013

    Videos de Experimentos Relacionados

    Last Updated: May 18, 2026

    A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia
    06:01

    A Thrombotic Stroke Model Based On Transient Cerebral Hypoxia-ischemia

    Published on: August 18, 2015

    Real-Time Monitoring and Modulation of Blood Pressure in a Rabbit Model of Ischemic Stroke
    09:00

    Real-Time Monitoring and Modulation of Blood Pressure in a Rabbit Model of Ischemic Stroke

    Published on: February 10, 2023

    2-Vessel Occlusion/Hypotension: A Rat Model of Global Brain Ischemia
    09:29

    2-Vessel Occlusion/Hypotension: A Rat Model of Global Brain Ischemia

    Published on: June 22, 2013