Jean Brachet (1909-1988) fue uno de los

Same author

[Cellular dynamics of pathogenesis induced by bovine leukemia virus].

Virologie (Montrouge, France)·2022

Same author

[From fundamental research to translational research in oncology].

Revue medicale de Liege·2021

Same author

Monitoring molecular response in adult T-cell leukemia by high-throughput sequencing analysis of HTLV-1 clonality.

Leukemia·2017

Same author

Evolution of microRNA expression during human bronchial squamous carcinogenesis.

The European respiratory journal·2008

Same author

The role of NPM, p14arf and MDM2 in precursors of bronchial squamous cell carcinoma.

The European respiratory journal·2008

Same author

HTLV-I infection of WE17/10 CD4+ cell line leads to progressive alteration of Ca2+ influx that eventually results in loss of CD7 expression and activation of an antiapoptotic pathway involving AKT and BAD which paves the way for malignant transformation.

Leukemia·2007

Videos de Experimentos Relacionados

Jean Brachet (1909-1988) fue uno de los

Videos de Conceptos Relacionados

[Cellular dynamics of pathogenesis induced by bovine leukemia virus].

[From fundamental research to translational research in oncology].

Monitoring molecular response in adult T-cell leukemia by high-throughput sequencing analysis of HTLV-1 clonality.

Evolution of microRNA expression during human bronchial squamous carcinogenesis.

The role of NPM, p14arf and MDM2 in precursors of bronchial squamous cell carcinoma.

HTLV-I infection of WE17/10 CD4+ cell line leads to progressive alteration of Ca2+ influx that eventually results in loss of CD7 expression and activation of an antiapoptotic pathway involving AKT and BAD which paves the way for malignant transformation.

Videos de Experimentos Relacionados

Jean Brachet (1909-1988) fue uno de los

Videos de Conceptos Relacionados

[Cellular dynamics of pathogenesis induced by bovine leukemia virus].

[From fundamental research to translational research in oncology].

Monitoring molecular response in adult T-cell leukemia by high-throughput sequencing analysis of HTLV-1 clonality.

Evolution of microRNA expression during human bronchial squamous carcinogenesis.

The role of NPM, p14arf and MDM2 in precursors of bronchial squamous cell carcinoma.

HTLV-I infection of WE17/10 CD4+ cell line leads to progressive alteration of Ca2+ influx that eventually results in loss of CD7 expression and activation of an antiapoptotic pathway involving AKT and BAD which paves the way for malignant transformation.

Videos de Experimentos Relacionados

Jean Brachet (1909-1988) fue uno de los

Videos de Conceptos Relacionados

Artículos Relacionados

[Cellular dynamics of pathogenesis induced by bovine leukemia virus].

[From fundamental research to translational research in oncology].

Monitoring molecular response in adult T-cell leukemia by high-throughput sequencing analysis of HTLV-1 clonality.

Evolution of microRNA expression during human bronchial squamous carcinogenesis.

The role of NPM, p14arf and MDM2 in precursors of bronchial squamous cell carcinoma.

HTLV-I infection of WE17/10 CD4+ cell line leads to progressive alteration of Ca2+ influx that eventually results in loss of CD7 expression and activation of an antiapoptotic pathway involving AKT and BAD which paves the way for malignant transformation.

Videos de Experimentos Relacionados

Jean Brachet (1909-1988) fue uno de los

Videos de Conceptos Relacionados

Artículos Relacionados

[Cellular dynamics of pathogenesis induced by bovine leukemia virus].

[From fundamental research to translational research in oncology].

Monitoring molecular response in adult T-cell leukemia by high-throughput sequencing analysis of HTLV-1 clonality.

Evolution of microRNA expression during human bronchial squamous carcinogenesis.

The role of NPM, p14arf and MDM2 in precursors of bronchial squamous cell carcinoma.

HTLV-I infection of WE17/10 CD4+ cell line leads to progressive alteration of Ca2+ influx that eventually results in loss of CD7 expression and activation of an antiapoptotic pathway involving AKT and BAD which paves the way for malignant transformation.