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Videos de Conceptos Relacionados

Acute Kidney Injury I: Introduction01:22

Acute Kidney Injury I: Introduction

478
Introduction:Acute Kidney Injury (AKI) describes a swift decrease in kidney function occurring over hours to days, characterized by the kidneys' failure to remove waste products from the bloodstream. This leads to dangerous complications like metabolic acidosis, fluid overload, and electrolyte imbalances, such as hyperkalemia, which can cause life-threatening arrhythmias. AKI is common in both hospital and outpatient settings, often triggered by dehydration, sepsis, or exposure to nephrotoxic...
478
Acute Kidney Injury IV: Diagnostic Studies and Prevention01:30

Acute Kidney Injury IV: Diagnostic Studies and Prevention

227
Accurate diagnosis and effective prevention are critical in managing Acute Kidney Injury (AKI), which is linked to high mortality rates ranging from 10% to 80%. Timely recognition of at-risk patients and careful monitoring can significantly reduce the likelihood of kidney damage.Diagnostic Assessments:The diagnostic process starts with a comprehensive medical history to identify prerenal, intrarenal, and postrenal causes.Prerenal causes, such as dehydration, hypotension, or blood loss, should...
227
Acute Kidney Injury II: Pathophysiology01:29

Acute Kidney Injury II: Pathophysiology

798
Acute kidney injury (AKI) causes are categorized into three primary categories based on the location of the injury: prerenal, intrarenal (or intrinsic), and postrenal causes. This classification guides clinical management and illustrates how different pathways can impair kidney function.Etiology and Pathophysiology of Acute Kidney Injury1. Prerenal causesEtiology: Prerenal Acute Kidney Injury, the most common type, occurs when reduced blood flow to the kidneys decreases filtration capacity...
798
Acute Kidney Injury III: Clinical Manifestations01:29

Acute Kidney Injury III: Clinical Manifestations

721
Acute Kidney Injury (AKI) progresses through distinct clinical phases: the oliguric, diuretic, and recovery phases, each marked by unique manifestations and challenges.Oliguric Phase:The oliguric phase is the initial stage of AKI, typically lasting 10 to 14 days. This phase is marked by a significant reduction in urine output, usually less than 400 mL per day, indicating decreased kidney function. Fluid retention is a prominent feature, leading to symptoms such as edema, hypertension, and...
721
Acute Kidney Injury V: Interprofessional Care01:20

Acute Kidney Injury V: Interprofessional Care

243
Acute Kidney Injury (AKI) requires a collaborative healthcare approach to restore renal function and prevent complications. Essential management strategies involve monitoring fluid and electrolyte balance, adjusting medications, initiating dialysis when necessary, and providing nutritional support.Fluid and Electrolyte ManagementFluid Monitoring: Regularly monitoring body weight, central venous pressure, and urine output helps detect fluid imbalances early. Patient intake and output are...
243
Acute Kidney Injury VI: Nursing Management01:22

Acute Kidney Injury VI: Nursing Management

327
Acute Kidney Injury (AKI) results in an inability to maintain fluid, electrolyte, and acid-base balance. Effective nursing management is critical in improving patient outcomes and includes comprehensive patient assessment and targeted interventions.Comprehensive Patient AssessmentA detailed history collection is essential, focusing on any recent infections, nephrotoxic medication use, or chronic conditions such as hypertension and diabetes that may contribute to AKI. During the physical...
327

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Year in review 2012: Critical Care--Nephrology.

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Healthcare systems and chronic kidney disease: putting the patient in control.

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Neutrophil gelatinase associated lipocalin in acute kidney injury.

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Potential use of biomarkers in acute kidney injury: report and summary of recommendations from the 10th Acute Dialysis Quality Initiative consensus conference.

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Video Experimental Relacionado

Updated: Jan 3, 2026

A Large Animal Model for Acute Kidney Injury by Temporary Bilateral Renal Artery Occlusion
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Lesiones renales agudas

Claudio Ronco1, Rinaldo Bellomo2, John A Kellum3

  • 1Department of Medicine, University of Padova, Padova, Italy; International Renal Research Institute of Vicenza, Vicenza, Italy; Department of Nephrology, San Bortolo Hospital, Vicenza, Italy.

Lancet (London, England)
|November 29, 2019
PubMed
Resumen

La lesión renal aguda (IRA) afecta al 10-15% de los pacientes hospitalizados y más del 50% en cuidados intensivos. Los nuevos biomarcadores e intervenciones son prometedores para el manejo de esta condición compleja.

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Área de la Ciencia:

  • Nefrología
  • Medicina de cuidados intensivos

Sus antecedentes:

  • La lesión renal aguda (IRA) se caracteriza por un rápido deterioro de la función renal, que afecta a una parte significativa de los pacientes hospitalizados y en estado crítico.
  • AKI puede progresar a enfermedad renal crónica, lo que pone de relieve la necesidad de detección y tratamiento tempranos.
  • La heterogeneidad de AKI, que abarca diversos síndromes como la sepsis y el síndrome cardiorrenal, complica el diagnóstico y el tratamiento.

Objetivo del estudio:

  • Proporcionar una visión general de la lesión renal aguda (IRA), incluyendo su definición, incidencia e implicaciones clínicas.
  • Discutir el papel de los biomarcadores emergentes en la evaluación de la lesión renal y la orientación de las estrategias terapéuticas.
  • Revisar los enfoques actuales para el manejo de la IAC, teniendo en cuenta el contexto clínico y la disponibilidad de recursos.

Principales métodos:

  • Revisión de la literatura de estudios recientes y directrices clínicas sobre la lesión renal aguda.
  • Análisis de la incidencia y prevalencia de la IAC en diferentes poblaciones de pacientes.
  • Evaluación del valor diagnóstico y pronóstico de los nuevos biomarcadores de la lesión renal.
  • Síntesis de las pruebas relativas a la eficacia de diversas intervenciones de AKI.

Principales resultados:

  • La incidencia de IAC es considerable, especialmente en las unidades de cuidados intensivos (UCI), donde supera el 50%.
  • Los biomarcadores ofrecen un potencial para mejorar la estratificación del riesgo y la terapia personalizada en la IAC.
  • A pesar de las controversias sobre el tratamiento, la evidencia creciente apoya el uso combinado de ciertas intervenciones.

Conclusiones:

  • La IAC es un síndrome complejo con una morbilidad y mortalidad significativas, que requiere estrategias de manejo adaptadas.
  • Los biomarcadores son cruciales para la detección temprana y la evaluación del riesgo, potencialmente orientando el tratamiento.
  • Si bien la eficacia del tratamiento varía, las intervenciones combinadas muestran una evidencia creciente de beneficio en el manejo de la IAC.