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Las mutaciones raras de FN1 indican un papel protector contra la demencia por cuerpos de Lewy en los portadores homocigotos de APOEε4
- Paolo Reho 1,2, Anindita Ray 1, Karri Kaivola 1,3, , Badri N Vardarajan 4,5, Haotian Wu 2, Sonja W Scholz 6,7
- Paolo Reho 1,2, Anindita Ray 1, Karri Kaivola 1,3
- 1Neurodegenerative Diseases Research Section, National Institute of Neurological Disorders and Stroke, Bethesda, MD, 20892-3707, USA.
- 2Department of Environmental Health Sciences, Mailman School of Public Health, Columbia University, New York, NY, USA.
- 3Translational Immunology, Research Programs Unit, University of Helsinki, Helsinki, Finland.
- 4Taub Institute for Research On Alzheimer's Disease and the Aging Brain, College of Physicians and Surgeons, Columbia University, New York, NY, USA.
- 5The Gertrude .H. Sergievsky Center, Vagelos College of Physicians and Surgeons, Columbia University, New York, NY, USA.
- 6Neurodegenerative Diseases Research Section, National Institute of Neurological Disorders and Stroke, Bethesda, MD, 20892-3707, USA. sonja.scholz@nih.gov.
- 7Department of Neurology, Johns Hopkins University Medical Center, Baltimore, MD, USA. sonja.scholz@nih.gov.
- 0Neurodegenerative Diseases Research Section, National Institute of Neurological Disorders and Stroke, Bethesda, MD, 20892-3707, USA.
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