Video Experimental Relacionado

Updated: May 12, 2026

Determining Immune System Suppression versus CNS Protection for Pharmacological Interventions in Autoimmune Demyelination

Determining Immune System Suppression versus CNS Protection for Pharmacological Interventions in Autoimmune Demyelination

Published on: September 12, 2016

Síndrome de Guillain-Barré precedido por la parálisis de Bell después de una gastroenteritis infecciosa aguda: un

Macy T Coleman¹, Nicholas G Boughton¹

¹Soldier Centered Medical Home-CAB, Desmond Doss Health Clinic, Wahiawa, Hawaii, HI 96786, United States.

Military medicine

|September 7, 2025

Resumen

El síndrome de Guillain-Barré (GBS), un trastorno inmunológico, puede presentarse inusualmente con debilidad en las extremidades superiores y problemas en los nervios craneales, no solo síntomas en las extremidades inferiores. El diagnóstico y el tratamiento tempranos son cruciales para la recuperación completa, especialmente para las personas activas.

Área de la Ciencia:

Neurología
Inmunología
Trastornos del sistema nervioso periférico

Sus antecedentes:

El síndrome de Guillain-Barré (GBS) es una poliradiculopatía inflamatoria aguda mediada por el sistema inmunológico.
El SGB se presenta típicamente con debilidad ascendente simétrica de las extremidades inferiores y disminución de los reflejos.
Las infecciones a menudo preceden a la aparición del SGB.

Objetivo del estudio:

Para reportar una presentación poco común de GBS en un piloto militar.
Para enfatizar las diversas manifestaciones clínicas de GBS.
Destacar la importancia de un diagnóstico y tratamiento oportunos del SGB para mantener la aptitud operativa.

Principales métodos:

Informe de caso de un piloto del ejército caucásico de 36 años.
Análisis detallado de la presentación clínica.
Revisión de los criterios de diagnóstico y los protocolos de tratamiento del SGB.

Principales resultados:

El paciente presentaba una presentación atípica de GBS que incluía neuropatía de las extremidades superiores y parálisis del nervio craneal.
Este caso subraya el variado espectro clínico de la EGB.
La intervención rápida facilitó la recuperación del paciente.

Conclusiones:

El SGB puede manifestarse con diversos déficits neurológicos más allá del patrón típico.
El diagnóstico precoz y preciso es vital para el tratamiento eficaz de la EGB.
El tratamiento exitoso del GBS es esencial para la recuperación del paciente y el retorno al servicio, incluido el estado de vuelo militar.

Más Videos Relacionados

Induction of Paralysis and Visual System Injury in Mice by T Cells Specific for Neuromyelitis Optica Autoantigen Aquaporin-4

Induction of Paralysis and Visual System Injury in Mice by T Cells Specific for Neuromyelitis Optica Autoantigen Aquaporin-4

Published on: August 21, 2017

A Simple Approach to Induce Experimental Autoimmune Neuritis in C57BL/6 Mice for Functional and Neuropathological Assessments

A Simple Approach to Induce Experimental Autoimmune Neuritis in C57BL/6 Mice for Functional and Neuropathological Assessments

Published on: November 9, 2017

Videos de Conceptos Relacionados

Diphtheria

Diphtheria

Diphtheria is an acute, toxin-mediated infectious disease that primarily affects the upper respiratory tract. It is caused by Corynebacterium diphtheriae, a Gram-positive, pleomorphic rod that lacks spore-forming capability and exhibits a characteristic club-shaped morphology under microscopic examination. While C. diphtheriae can asymptomatically colonize mucosal surfaces, clinical disease manifests only when the bacterial strain is lysogenized by a specific β-corynephage. This phage...

Bacterial Gastroenteritis

Bacterial Gastroenteritis

Bacterial gastroenteritis, characterized by diarrhea, abdominal cramps, and vomiting, is often caused by ingestion of contaminated food or water and is frequently associated with pathogenic Escherichia coli strains. These microbes exploit two principal mechanisms to inflict disease.Shiga toxin–producing E. coli, also referred to as STEC—notably O157:H7—release Shiga toxins that target ribosomes, blocking protein synthesis. The B subunit of the toxin binds the host glycolipid receptor...

Viral Meningitis

Viral Meningitis

Viral meningitis is the most common form of meningitis and is often referred to as aseptic meningitis to indicate the absence of bacterial involvement. It is generally milder than bacterial meningitis, with symptoms including fever, headache, stiff neck, drowsiness, nausea, photophobia, and vomiting. Rarely, more severe manifestations or death may occur. Common causative agents include enteroviruses, particularly coxsackie A and B viruses and echoviruses, all members of the Enterovirus genus...

Bacterial Meningitis I: Introduction

Bacterial Meningitis I: Introduction

Bacterial meningitis is a severe, life-threatening inflammation of the meninges, particularly the pia mater and arachnoid mater, affecting the subarachnoid space, ventricles, and cerebrospinal fluid (CSF). If untreated, it can lead to significant neurological complications or death.Causative AgentsCommon pathogens vary with age and immune status. In adults, major organisms include Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. Streptococcus agalactiae (group B...

Encephalitis l: Introduction

Encephalitis l: Introduction

Encephalitis is inflammation of the brain parenchyma, most often due to infections or autoimmune processes. It presents with neuropsychiatric features such as fever, altered mental status, behavioral changes, cognitive dysfunction, seizures, focal deficits, and sometimes autonomic instability. In some cases, the meninges are also involved, resulting in meningoencephalitis.Infectious CausesInfectious encephalitis is most commonly viral but can also result from bacterial, fungal, or parasitic...

Encephalitis ll: Pathophysiology

Encephalitis ll: Pathophysiology

Encephalitis is inflammation of the brain parenchyma caused by direct viral invasion or immune-mediated mechanisms triggered by infections or tumors. Both processes lead to neuronal injury, disrupted neurotransmission, and diverse neurological symptoms, often with overlapping clinical and pathological features.Autoimmune EncephalitisIn autoimmune encephalitis, antibodies target neuronal antigens on cell surfaces, synapses, or within neurons. A key example is anti-NMDAR encephalitis, which can...