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Intersecciones GM1 diseñadas entre las vías mitocondriales y sinápticas para mejorar la patología de la ELA

Federica Pilotto1,2, Tristan Dellazizzo Toth3,4, Silvano Bond3,4

  • 1Institut Neuromyogène, Pathophysiology and genetics of the neuron and muscle, Inserm U1315, CNRS, Université Claude Bernard Lyon I, Lyon, France.

Advanced science (Weinheim, Baden-Wurttemberg, Germany)
|January 5, 2026
PubMed
Resumen

No abstract available in PubMed .

Palabras clave:
No obstante, la Comisión no ha adoptado ninguna decisión al respecto.Contactos entre las ER y las mitocondriasGM1 y GM2En el caso de los vehículos de pasajeros, se aplicará el siguiente procedimiento:Esclerosis lateral amiotrófica (ELA)Disfunción mitocondrialformulación nanoliposomalProteomíaDisfunción sináptica

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