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Acute Kidney Injury IV: Diagnostic Studies and Prevention01:30

Acute Kidney Injury IV: Diagnostic Studies and Prevention

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Accurate diagnosis and effective prevention are critical in managing Acute Kidney Injury (AKI), which is linked to high mortality rates ranging from 10% to 80%. Timely recognition of at-risk patients and careful monitoring can significantly reduce the likelihood of kidney damage.Diagnostic Assessments:The diagnostic process starts with a comprehensive medical history to identify prerenal, intrarenal, and postrenal causes.Prerenal causes, such as dehydration, hypotension, or blood loss, should...
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Acute Kidney Injury V: Interprofessional Care01:20

Acute Kidney Injury V: Interprofessional Care

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Acute Kidney Injury (AKI) requires a collaborative healthcare approach to restore renal function and prevent complications. Essential management strategies involve monitoring fluid and electrolyte balance, adjusting medications, initiating dialysis when necessary, and providing nutritional support.Fluid and Electrolyte ManagementFluid Monitoring: Regularly monitoring body weight, central venous pressure, and urine output helps detect fluid imbalances early. Patient intake and output are...
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Acute Kidney Injury II: Pathophysiology01:29

Acute Kidney Injury II: Pathophysiology

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Acute kidney injury (AKI) causes are categorized into three primary categories based on the location of the injury: prerenal, intrarenal (or intrinsic), and postrenal causes. This classification guides clinical management and illustrates how different pathways can impair kidney function.Etiology and Pathophysiology of Acute Kidney Injury1. Prerenal causesEtiology: Prerenal Acute Kidney Injury, the most common type, occurs when reduced blood flow to the kidneys decreases filtration capacity...
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Acute Kidney Injury I: Introduction01:22

Acute Kidney Injury I: Introduction

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Introduction:Acute Kidney Injury (AKI) describes a swift decrease in kidney function occurring over hours to days, characterized by the kidneys' failure to remove waste products from the bloodstream. This leads to dangerous complications like metabolic acidosis, fluid overload, and electrolyte imbalances, such as hyperkalemia, which can cause life-threatening arrhythmias. AKI is common in both hospital and outpatient settings, often triggered by dehydration, sepsis, or exposure to nephrotoxic...
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Acute Kidney Injury III: Clinical Manifestations01:29

Acute Kidney Injury III: Clinical Manifestations

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Acute Kidney Injury (AKI) progresses through distinct clinical phases: the oliguric, diuretic, and recovery phases, each marked by unique manifestations and challenges.Oliguric Phase:The oliguric phase is the initial stage of AKI, typically lasting 10 to 14 days. This phase is marked by a significant reduction in urine output, usually less than 400 mL per day, indicating decreased kidney function. Fluid retention is a prominent feature, leading to symptoms such as edema, hypertension, and...
802
Acute Kidney Injury VI: Nursing Management01:22

Acute Kidney Injury VI: Nursing Management

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Acute Kidney Injury (AKI) results in an inability to maintain fluid, electrolyte, and acid-base balance. Effective nursing management is critical in improving patient outcomes and includes comprehensive patient assessment and targeted interventions.Comprehensive Patient AssessmentA detailed history collection is essential, focusing on any recent infections, nephrotoxic medication use, or chronic conditions such as hypertension and diabetes that may contribute to AKI. During the physical...
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Cushing's syndrome and early growth hormone hypersecretion in a child with Carney complex: a case report.

Frontiers in endocrinology·2026
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Uric acid to high density lipoprotein-cholesterol ratio as a potential biomarker of comorbidities in youth with obesity.

Nutrition, metabolism, and cardiovascular diseases : NMCD·2026
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Advancing Insights into Biomarkers in Congenital Anomalies of the Kidney and Urinary Tract: A Scoping Review.

Cells·2026
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Prevalence and factors associated with acute kidney injury in children presenting to the emergency department with a first tonic-clonic seizure: an exploratory study.

European journal of pediatrics·2026
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Glomerular hyperfiltration and increased peripheral sensitivity to thyroid hormones in youth with overweight or obesity.

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Vitamin D and Mean Platelet Volume as Biomarkers in Pediatric Obstructive Sleep Apnea: Associations with Disease Severity and Sleep Parameters.

Journal of clinical medicine·2026

Video Experimental Relacionado

Updated: Jan 13, 2026

Unilateral Ureteral Obstruction Model for Investigating Kidney Interstitial Fibrosis
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Unilateral Ureteral Obstruction Model for Investigating Kidney Interstitial Fibrosis

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Lesión Renal Aguda en Niños con Poliuria: Una Revisión Sistemática

Giulio Rivetti1, Mariantonia Braile1, Anna Di Sessa1

  • 1Department of Woman, Child and of General and Specialized Surgery, Università Degli Studi Della Campania "Luigi Vanvitelli", Via Luigi De Crecchio 2, 80138 Naples, Italy.

Journal of clinical medicine
|January 10, 2026
PubMed
Resumen

La lesión renal aguda (LRA) puede presentarse con poliuria en niños, particularmente en casos de cetoacidosis diabética (CAD) y otras afecciones. Esta revisión sintetiza los hallazgos sobre las presentaciones de LRA y poliuria pediátricas.

Palabras clave:
LRAlesión renal agudaniñosdiagnósticopediátricopoliuria

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Author Spotlight: Developing a Bedside Protocol for Kidney and Genitourinary Ultrasonography
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Área de la Ciencia:

  • Nefrología Pediátrica
  • Medicina de Cuidados Críticos
  • Endocrinología

Sus antecedentes:

  • La lesión renal aguda (LRA) y la poliuria en niños presentan desafíos diagnósticos y de manejo.
  • Comprender la interacción entre la LRA y la poliuria es crucial para la atención del paciente pediátrico.

Objetivo del estudio:

  • Revisar y sintetizar sistemáticamente las presentaciones de LRA en niños con poliuria.
  • Identificar las afecciones subyacentes asociadas con LRA y poliuria concurrentes en poblaciones pediátricas.

Principales métodos:

  • Búsqueda sistemática de literatura en las bases de datos Embase, PubMed y Scopus.
  • Inclusión de estudios e informes de casos centrados en LRA y poliuria pediátricas.
  • Evaluación de la calidad metodológica de las publicaciones seleccionadas.

Principales resultados:

  • Se analizaron datos de 32 pacientes pediátricos (edad media 11,02 años).
  • La poliuria estuvo presente en 26 pacientes, siendo la cetoacidosis diabética (CAD) la afección asociada más común (19 casos).
  • De los 20 pacientes con poliuria y datos disponibles, 9 (45%) desarrollaron LRA, y varios alcanzaron el estadio 3 de KDIGO.

Conclusiones:

  • La poliuria puede ser parte integral de la fisiopatología de la LRA en niños.
  • Se observó una relación entre la poliuria y la LRA significativa (estadio KDIGO ≥ 2) en trastornos metabólicos como la CAD, afecciones nefrológicas como el síndrome de Bartter y casos oncológicos como el neuroblastoma.