Jove
Visualize
Contáctanos
JoVE
x logofacebook logolinkedin logoyoutube logo
ACERCA DE JoVE
Visión GeneralLiderazgoBlogCentro de Ayuda JoVE
AUTORES
Proceso de PublicaciónConsejo EditorialAlcance y PolíticasRevisión por ParesPreguntas FrecuentesEnviar
BIBLIOTECARIOS
TestimoniosSuscripcionesAccesoRecursosConsejo Asesor de BibliotecasPreguntas Frecuentes
INVESTIGACIÓN
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchivo
EDUCACIÓN
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualCentro de Recursos para ProfesoresSitio de Profesores
Términos y Condiciones de Uso
Política de Privacidad
Políticas

Videos de Conceptos Relacionados

Nephrotic Syndrome I : Introduction01:24

Nephrotic Syndrome I : Introduction

609
Nephrotic Syndrome is a chronic kidney disorder defined by clinical findings such as severe proteinuria, hypoalbuminemia, hyperlipidemia, and edema. These symptoms result from damage to the glomeruli, the kidney’s filtering units, increasing their permeability to proteins.Definition and Meaning:Proteinuria, defined as the loss of more than 3.5 grams of protein per day in adults, is a crucial feature of nephrotic syndrome. This condition is often accompanied by edema, the accumulation of...
609
Venous Thrombosis I: Introduction01:30

Venous Thrombosis I: Introduction

433
Venous thrombosis, the most common disorder of the veins, involves the formation of a thrombus or blood clot associated with vein inflammation. It can be classified as either superficial vein thrombosis or deep vein thrombosis.Superficial Vein Thrombosis: This involves the formation of a thrombus in a superficial vein, usually the greater or lesser saphenous vein. Though less severe than deep vein thrombosis (DVT), SVT can lead to complications if untreated.Deep Vein Thrombosis (DVT): This...
433
Nephrotic Syndrome II : Assessment and Medical Management01:26

Nephrotic Syndrome II : Assessment and Medical Management

237
IntroductionNephrotic syndrome is a kidney disorder marked by excessive protein loss in the urine, leading to various systemic complications. This condition often results from damage to the glomeruli—the kidney's filtering units—causing proteinuria, low blood protein levels, and fluid retention. Understanding the assessment, diagnosis, and management of nephrotic syndrome is essential for effective treatment and prevention of further kidney damage.AssessmentPatient History: Document...
237
Nephrotic Syndrome III : Nursing Management01:24

Nephrotic Syndrome III : Nursing Management

317
Nursing management for nephrotic syndrome adapts as the disease progresses, with strategies evolving to address advancing symptoms and complications.Early-Stage Management In the early stages, nursing interventions for nephrotic syndrome resemble those used in managing acute glomerulonephritis, focusing on symptom monitoring, fluid balance, and managing mild to moderate edema.Vital Signs: Regularly monitor blood pressure, pulse, respiratory rate, and temperature to promptly identify...
317
Venous Thrombosis III: Interprofessional Care01:29

Venous Thrombosis III: Interprofessional Care

307
Venous thrombosis requires effective prevention and treatment strategies to improve patient outcomes and reduce potential complications.Prevention StrategiesHealthcare providers must prioritize preventing venous thromboembolism (VTE) for all adult patients upon admission. Interventions depend on bleeding and thrombosis risk, medical history, current medications, diagnoses, planned procedures, and patient preferences. Patients on bed rest should change positions every two hours and, if not...
307
Venous Thrombosis IV: Nursing Management01:30

Venous Thrombosis IV: Nursing Management

242
Nursing management begins with a thorough assessment of the patient's health history. Key factors include trauma to veins, peripherally inserted central catheters, varicose veins, recent pregnancy or childbirth, surgery, bacteremia, prolonged bed rest, atrial fibrillation, COPD, heart failure, cancer, coagulation disorders, myocardial infarction, spinal cord injury, stroke, prolonged travel, recent bone fractures, and dehydration. Review medication intake, particularly oral contraceptives,...
242

También podría leer

Artículos Relacionados

Artículos vinculados a este trabajo por autores compartidos, revista y gráfico de citas.

Ordenar por
Same author

Metastatic Malignant Melanoma Masquerading as Tolosa-Hunt Syndrome.

Case reports in ophthalmological medicine·2026
Same author

Redefining Obstructive Sleep Apnea: Treatment in the Modern Era.

Pathophysiology : the official journal of the International Society for Pathophysiology·2026
Same author

Cytomegalovirus Lymphadenitis Mimicking Relapsed Lymphoma After CD19 Chimeric Antigen Receptor (CAR) T Cell Therapy.

APMIS : acta pathologica, microbiologica, et immunologica Scandinavica·2026
Same author

From Spider Bite to Fungating Ulcerating Mass: An Aggressive Squamous Cell Carcinoma of the Face.

Cureus·2025
Same author

Fragmented Chemotherapy of Plasmablastic Lymphoma in an Immunocompromised Patient.

Cureus·2025
Same author

Post-Hospitalization Home Monitoring Programs During the COVID-19 Pandemic: Survey Results from the Hospital Medicine Re-engineering Network (HOMERuN).

Journal of general internal medicine·2023

Video Experimental Relacionado

Updated: Jan 29, 2026

Induction of Nephrotic Syndrome in Mice by Retrobulbar Injection of Doxorubicin and Prevention of Volume Retention by Sustained Release Aprotinin
07:38

Induction of Nephrotic Syndrome in Mice by Retrobulbar Injection of Doxorubicin and Prevention of Volume Retention by Sustained Release Aprotinin

Published on: May 6, 2018

8.8K

Trombosis a pesar de proteinuria subnefrótica

Jose Redondo1, Alexander J Dobek1, Leonardo B Sosa1

  • 1Internal Medicine, University of Miami Miller School of Medicine, Jackson Memorial Hospital, Miami, USA.

Cureus
|January 28, 2026
PubMed
Resumen
Este resumen es generado por máquina.

Los pacientes con proteinuria subnefrótica y albúmina baja pueden enfrentar altos riesgos de coagulación, similares al síndrome nefrótico. Esto resalta la necesidad de un manejo cuidadoso del tromboembolismo venoso (TEV) en estas personas.

Palabras clave:
amiloidosis aahipoalbuminemiaanticoagulación peri-procedimientoproteinuria subnefróticatromboembolismo venoso (tev)

Más Videos Relacionados

Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models
04:40

Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models

Published on: January 5, 2024

3.2K
Ferric Chloride-induced Murine Thrombosis Models
10:37

Ferric Chloride-induced Murine Thrombosis Models

Published on: September 5, 2016

23.1K

Videos de Experimentos Relacionados

Last Updated: Jan 29, 2026

Induction of Nephrotic Syndrome in Mice by Retrobulbar Injection of Doxorubicin and Prevention of Volume Retention by Sustained Release Aprotinin
07:38

Induction of Nephrotic Syndrome in Mice by Retrobulbar Injection of Doxorubicin and Prevention of Volume Retention by Sustained Release Aprotinin

Published on: May 6, 2018

8.8K
Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models
04:40

Author Spotlight: Advancing Cancer Associated Thrombosis Research in Rodent Models

Published on: January 5, 2024

3.2K
Ferric Chloride-induced Murine Thrombosis Models
10:37

Ferric Chloride-induced Murine Thrombosis Models

Published on: September 5, 2016

23.1K

Área de la Ciencia:

  • Nefrología
  • Hematología
  • Medicina Interna

Sus antecedentes:

  • El síndrome nefrótico es un factor de riesgo conocido para el tromboembolismo venoso (TEV).
  • La proteinuria subnefrótica y la hipoalbuminemia también son reconocidas cada vez más como contribuyentes a la hipercoagulabilidad.
  • La comprensión del riesgo de coagulación en estas afecciones es crucial para el manejo del paciente.

Objetivo del estudio:

  • Destacar la asociación entre la proteinuria subnefrótica, la hipoalbuminemia y un mayor riesgo de TEV.
  • Presentar un caso que ilustra el potencial de desarrollo de TEV incluso cuando la anticoagulación se interrumpe temporalmente.
  • Enfatizar la necesidad de un enfoque integral para el manejo del riesgo de TEV en pacientes con enfermedad renal.

Principales métodos:

  • Informe de caso de un paciente de 40 años que presenta anasarca, disnea y trombosis venosa profunda (TVP).
  • Observación clínica del desarrollo de TEV durante la interrupción temporal de la anticoagulación antes de una biopsia renal.
  • Revisión de la presentación clínica y los hallazgos diagnósticos del paciente.

Principales resultados:

  • El paciente presentó TVP bilaterales en extremidades inferiores y embolia pulmonar.
  • Se desarrollaron nuevas TVP bilaterales en extremidades superiores tras la interrupción temporal de la anticoagulación.
  • Se observó hipoalbuminemia en el contexto de proteinuria subnefrótica como un posible indicador de riesgo de coagulación.

Conclusiones:

  • La proteinuria subnefrótica y la hipoalbuminemia justifican la consideración como factores de riesgo significativos para el TEV.
  • Son esenciales estrategias de manejo cuidadosas para los pacientes con estas afecciones, particularmente cuando los procedimientos requieren la interrupción temporal de la anticoagulación.
  • Se recomienda un enfoque multidisciplinario para optimizar la prevención y el tratamiento del TEV en esta población de pacientes.