Cambio de conmutación de la hemoglobina.

Same author

Lentiviral vectors for the treatment of Wiskott-Aldrich syndrome.

Gene therapy·2008

Same author

Current status and prospects for gene therapy.

Vox sanguinis·2004

Same author

Functional correction of T cells derived from patients with the Wiskott-Aldrich syndrome (WAS) by transduction with an oncoretroviral vector encoding the WAS protein.

Gene therapy·2003

Same author

Bone marrow-derived cells contribute to tumor neovasculature and, when modified to express an angiogenesis inhibitor, can restrict tumor growth in mice.

Clinical cancer research : an official journal of the American Association for Cancer Research·2001

Same author

Autocrine expression of both endostatin and green fluorescent protein provides a synergistic antitumor effect in a murine neuroblastoma model.

Cancer gene therapy·2001

Same author

Functional requirements for phenotypic correction of murine beta-thalassemia: implications for human gene therapy.

Blood·2001

Videos de Experimentos Relacionados

Cambio de conmutación de la hemoglobina.

Videos de Conceptos Relacionados

Lentiviral vectors for the treatment of Wiskott-Aldrich syndrome.

Current status and prospects for gene therapy.

Functional correction of T cells derived from patients with the Wiskott-Aldrich syndrome (WAS) by transduction with an oncoretroviral vector encoding the WAS protein.

Bone marrow-derived cells contribute to tumor neovasculature and, when modified to express an angiogenesis inhibitor, can restrict tumor growth in mice.

Autocrine expression of both endostatin and green fluorescent protein provides a synergistic antitumor effect in a murine neuroblastoma model.

Functional requirements for phenotypic correction of murine beta-thalassemia: implications for human gene therapy.

Videos de Experimentos Relacionados

Cambio de conmutación de la hemoglobina.

Videos de Conceptos Relacionados

Lentiviral vectors for the treatment of Wiskott-Aldrich syndrome.

Current status and prospects for gene therapy.

Functional correction of T cells derived from patients with the Wiskott-Aldrich syndrome (WAS) by transduction with an oncoretroviral vector encoding the WAS protein.

Bone marrow-derived cells contribute to tumor neovasculature and, when modified to express an angiogenesis inhibitor, can restrict tumor growth in mice.

Autocrine expression of both endostatin and green fluorescent protein provides a synergistic antitumor effect in a murine neuroblastoma model.

Functional requirements for phenotypic correction of murine beta-thalassemia: implications for human gene therapy.

Videos de Experimentos Relacionados

Cambio de conmutación de la hemoglobina.

Videos de Conceptos Relacionados

Artículos Relacionados

Lentiviral vectors for the treatment of Wiskott-Aldrich syndrome.

Current status and prospects for gene therapy.

Functional correction of T cells derived from patients with the Wiskott-Aldrich syndrome (WAS) by transduction with an oncoretroviral vector encoding the WAS protein.

Bone marrow-derived cells contribute to tumor neovasculature and, when modified to express an angiogenesis inhibitor, can restrict tumor growth in mice.

Autocrine expression of both endostatin and green fluorescent protein provides a synergistic antitumor effect in a murine neuroblastoma model.

Functional requirements for phenotypic correction of murine beta-thalassemia: implications for human gene therapy.

Videos de Experimentos Relacionados

Cambio de conmutación de la hemoglobina.

Videos de Conceptos Relacionados

Artículos Relacionados

Lentiviral vectors for the treatment of Wiskott-Aldrich syndrome.

Current status and prospects for gene therapy.

Functional correction of T cells derived from patients with the Wiskott-Aldrich syndrome (WAS) by transduction with an oncoretroviral vector encoding the WAS protein.

Bone marrow-derived cells contribute to tumor neovasculature and, when modified to express an angiogenesis inhibitor, can restrict tumor growth in mice.

Autocrine expression of both endostatin and green fluorescent protein provides a synergistic antitumor effect in a murine neuroblastoma model.

Functional requirements for phenotypic correction of murine beta-thalassemia: implications for human gene therapy.