:

N G Pérez1, K Hashimoto, S McCune

  • 1Section of Molecular and Cellular Cardiology, Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Md, USA.

Circulation
|March 2, 1999
PubMed
まとめ

心不全は,ミオフィラメントの活性化がひどく鈍化した状態である. 変化した細胞内カルシウム (Ca2+) 運動は,この収縮機構の機能不全を補償し,現在の心不全の病理生理学概念に挑戦します.

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