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Updated: Jul 5, 2026

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ドナーの心臓における心筋機能不全. 可能性のある病因学

V J Owen1, P B Burton, M C Michel

  • 1Cardiothoracic Surgery, National Heart and Lung Institute at Imperial College School of Medicine, London, UK. v.j.owen@ic.ac.uk

Circulation
|May 20, 1999
PubMed
まとめ

抑制性Gタンパク質ギアルファ活性の増加は,急性欠陥のドナー心臓の収縮性の低下に寄与する. これは,カルシウム処理タンパク質とβ-アドレノ受容体の密度の変化と対照的であるが,これらは一般的な原因ではなかった.

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