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Updated: Jan 18, 2026

Implementation of a Real-Time Psychosis Risk Detection and Alerting System Based on Electronic Health Records using CogStack
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集積された低密度リポプロテインの吸収は,膜組織因子のプロコアグラント活性と,人間の血管の滑らかな筋肉細胞におけるマイクロ粒子の放出を誘導する

Vicenta Llorente-Cortés1, Marta Otero-Viñas, Sandra Camino-López

  • 1Cardiovascular Research Center, CSIC-ICCC, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

Circulation
|July 9, 2004
PubMed
まとめ

血管の滑らかな筋肉細胞 (VSMC) によって集積されたLDL (agLDL) の内部化は,組織因子 (TF) の血栓活性を引き起こす. このagLDL誘発のTF放出は,LDL受容体に関連するタンパク質によって媒介され,アポトーシスとは関係ありません.

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