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Updated: Jul 17, 2026

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アデノシンA2A受容体活性化の心筋梗塞を抑制する効果は,CD4+Tリンパ球に対する作用によるものです

Zequan Yang1, Yuan-Ji Day, Marie-Claire Toufektsian

  • 1Department of Biomedical Engineering, University of Virginia Health System, Box 800759, Charlottesville, VA 22903, USA. zy6b@virginia.edu

Circulation
|October 25, 2006
PubMed
まとめ

アデノシンA2A受容体 (A2AR) の活性化により,CD4+T細胞を抑制することにより,心臓発作の損傷を軽減します. この研究では,CD4+Tリンパ球が,心筋不全/再流血損傷の主要な要因であることを明らかにしました.

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