付属経路でリンクする. アンテグラード前刺激の機能的喪失
M D Gonzalez1, A J Greenspon, G A Kidwell
1Department of Medicine, Jefferson Medical College, Thomas Jefferson University, Philadelphia, PA.
Circulation
|April 1, 1991
まとめ
ウォルフ・パーキンソン・ホワイト症候群の隠された逆行活性化,またはリンクは,付属経路の双方向ブロックを伴う. この現象は,反射性および逆行活性化遅延に依存し,心室外刺激によって中断されることがあります.
科学分野:
- 電気生理学 電気生理学
- 心臓病学 心臓病学
背景:
- 隠された逆行活性化 (concealed retrograde activation) は,アンテグラード伝導ブロックのための提案されたメカニズムである.
- この研究では,ウォルフ・パーキンソン・ホワイト症候群の患者10人の関連性を調査した.
研究 の 目的:
- 隠された逆行アクティベーションがアンテグラード伝導ブロックを引き起こすという仮説を検証するために.
- 付属経路の接続条件と特徴を定義する.
主な方法:
- ウォルフ・パーキンソン・ホワイト症候群の患者は,過剰な動力による心房ペースを施して,持続的なアンテグレイド・ブロックを誘発した.
- atrial pacingプロトコルは,サイクル長さの"リンクウィンドウ"を定義しました.
- 早期の心室外刺激は,結合を中断するために使用されました.
主要な成果:
- リンクウィンドウ (70~290 msec) が特定され,アクセサリー経路の伝導は前の状態に依存していた.
- リンクウィンドウは,逆行的なアクティベーションの遅延を推定した.
- アクセサリー経路の反射性は,心房内側経路の反射性を上回り,心房外刺激によって中断される可能性があります.
結論:
- 付属経路のリンクは,双方向ブロックと関連しています.
- リンクの開始と安定性は,アンテグレイド・リフレクター性とレトログラード・アクティベーション遅延の関係に依存する.
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