スナップショット: 細胞体の細胞体

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Patient-derived organoid xenografts reveal the multifaceted role of the lncRNA <i>MALAT1</i> in breast cancer progression.

bioRxiv : the preprint server for biology·2026

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A sub-set of guanine- and cytosine-rich genes are actively transcribed at the nuclear lamin B1 region.

Cellular and molecular life sciences : CMLS·2026

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Longitudinal tracking of <i>MALAT1</i> level over a breast cancer patient's course of treatment and disease progression.

Molecular therapy. Oncology·2025

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Loss of BPTF restores estrogen response and suppresses metastasis of mammary tumors.

Nature communications·2025

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On-target toxicity limits the efficacy of CDK11 inhibition against cancers with 1p36 deletions.

bioRxiv : the preprint server for biology·2025

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LINC01235 Is an Upstream Regulator of the NFIB Gene and the Notch Pathway in Triple-Negative Breast Cancer.

Molecular cancer research : MCR·2025

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Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.

Cell·2026

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Systematic discovery of pathogen effector functions across human pathogens and pathways.

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Structural basis for host membrane binding and remodeling by invading malaria parasites.

Cell·2026

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Multiscale integration of tissue and chromatin context converts cell heterogeneity into stable intestinal patterning.

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Arc mediates intercellular tau transmission via extracellular vesicles.

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Electromagnetic field-inducible in vivo gene switch for remote spatiotemporal control of gene expression.

Cell·2026

関連する実験動画

スナップショット: 細胞体の細胞体

関連する概念動画

Patient-derived organoid xenografts reveal the multifaceted role of the lncRNA <i>MALAT1</i> in breast cancer progression.

A sub-set of guanine- and cytosine-rich genes are actively transcribed at the nuclear lamin B1 region.

Longitudinal tracking of <i>MALAT1</i> level over a breast cancer patient's course of treatment and disease progression.

Loss of BPTF restores estrogen response and suppresses metastasis of mammary tumors.

On-target toxicity limits the efficacy of CDK11 inhibition against cancers with 1p36 deletions.

LINC01235 Is an Upstream Regulator of the NFIB Gene and the Notch Pathway in Triple-Negative Breast Cancer.

Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.

Systematic discovery of pathogen effector functions across human pathogens and pathways.

Structural basis for host membrane binding and remodeling by invading malaria parasites.

Multiscale integration of tissue and chromatin context converts cell heterogeneity into stable intestinal patterning.

Arc mediates intercellular tau transmission via extracellular vesicles.

Electromagnetic field-inducible in vivo gene switch for remote spatiotemporal control of gene expression.

関連する実験動画

スナップショット: 細胞体の細胞体

関連する概念動画

関連記事

Patient-derived organoid xenografts reveal the multifaceted role of the lncRNA <i>MALAT1</i> in breast cancer progression.

A sub-set of guanine- and cytosine-rich genes are actively transcribed at the nuclear lamin B1 region.

Longitudinal tracking of <i>MALAT1</i> level over a breast cancer patient's course of treatment and disease progression.

Loss of BPTF restores estrogen response and suppresses metastasis of mammary tumors.

On-target toxicity limits the efficacy of CDK11 inhibition against cancers with 1p36 deletions.

LINC01235 Is an Upstream Regulator of the NFIB Gene and the Notch Pathway in Triple-Negative Breast Cancer.

Whole-cell particle-based digital twin simulations from 4D lattice light-sheet microscopy data.

Systematic discovery of pathogen effector functions across human pathogens and pathways.

Structural basis for host membrane binding and remodeling by invading malaria parasites.

Multiscale integration of tissue and chromatin context converts cell heterogeneity into stable intestinal patterning.

Arc mediates intercellular tau transmission via extracellular vesicles.

Electromagnetic field-inducible in vivo gene switch for remote spatiotemporal control of gene expression.