p90rskによるErp1のリン酸化は,Xenopus laevisの卵における細胞静止因子停止のために必要である

Tomoko Nishiyama1, Keita Ohsumi, Takeo Kishimoto

  • 1Laboratory of Cell and Developmental Biology, Graduate School of Bioscience and Biotechnology, Tokyo Institute of Technology, 4259 Nagatsuta, Midori-ku, Yokohama 226-8501, Japan.

Nature
|April 6, 2007
PubMed
まとめ

MosとErp1/Emi2は,Xenopusの卵における細胞静止因子 (CSF) の停止を維持するために協力しています. モスに依存したリン酸化はErp1/Emi2を安定させ,アナフェーズ促進複合体/サイクロソーム (APC/C) の抑制を強化する.

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