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Updated: May 10, 2026

Enhancement of Apoptotic and Autophagic Induction by a Novel Synthetic C-1 Analogue of 7-deoxypancratistatin in Human Breast Adenocarcinoma and Neuroblastoma Cells with Tamoxifen
19:44

Enhancement of Apoptotic and Autophagic Induction by a Novel Synthetic C-1 Analogue of 7-deoxypancratistatin in Human Breast Adenocarcinoma and Neuroblastoma Cells with Tamoxifen

Published on: May 30, 2012

腫瘍抑制剤Par-4は,アポトーシスの外部経路を活性化させます

Ravshan Burikhanov1, Yanming Zhao, Anindya Goswami

  • 1Department of Radiation Medicine, University of Kentucky, Lexington, KY 40536, USA.

Cell
|July 28, 2009
PubMed
まとめ

前立腺アポトーシス反応-4 (Par-4) は細胞によって分泌され,細胞表面のGRP78.8に結合することによって癌細胞アポトーシスを誘導します. この細胞外Par-4シグナリングは,死の経路を活性化し,潜在的な治療目標を提供します.

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Fluorescence-based Monitoring of PAD4 Activity via a Pro-fluorescence Substrate Analog08:37

Fluorescence-based Monitoring of PAD4 Activity via a Pro-fluorescence Substrate Analog

Published on: November 5, 2014

In Vivo Inhibition of MicroRNA to Decrease Tumor Growth in Mice07:02

In Vivo Inhibition of MicroRNA to Decrease Tumor Growth in Mice

Published on: August 23, 2019

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