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Updated: May 5, 2026

Hyperinsulinemic-euglycemic Clamps in Conscious, Unrestrained Mice
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プロリルヒドロキシラーゼドメインタンパク質2は,食事による肥満およびグルコース不耐性において重要な役割を果たします

Hirohide Matsuura1, Toshihiro Ichiki, Eriko Inoue

  • 1Department of Cardiovascular Medicine, Kyushu University Graduate School of Medical Sciences, Fukuoka, Japan.

Circulation
|May 1, 2013
PubMed
まとめ

脂肪細胞内のプロリルヒドロキシラーゼドメインタンパク質2 (PHD2) の削除は,食事による肥満を予防し,グルコース代謝を改善します. これはPHD2を強調しています.

キーワード:
アディポサイトはアディポサイトです.細胞の低酸素症は肥満 肥満 肥満プロリルヒドロキシラーゼドメインタンパク質

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