D M Kerins1, L Roy, G A FitzGerald

  • 1Division of Clinical Pharmacology, Vanderbilt University, Nashville, Tennessee 37232.

Circulation
|December 1, 1989
PubMed
まとめ

血小板活性化は,組織型プラズミノゲン活性化剤 (t-PA) による冠動脈血栓溶解中に増加します. アスピリンはこれを阻害し,再注入された患者のプロスタサイクリン (PGI2) の減少は血管損傷の兆候となる可能性があります.

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