HipBA ,

Maria A Schumacher1, Pooja Balani2, Jungki Min1

  • 1Department of Biochemistry, Duke University School of Medicine, Durham, North Carolina 27710, USA.

Nature
|July 30, 2015
PubMed
まとめ

慢性感染症における多剤耐性は 休眠状態の持続細胞から生じる. 新しい研究により HipAタンパク質の二分化が この抗生物質耐性を制御し 尿路感染症の 洞察力を提供していることが明らかになりました

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