,寿

Scott F Leiser1, Hillary Miller1, Ryan Rossner1

  • 1Department of Pathology, University of Washington, Seattle, WA 98195, USA.

Science (New York, N.Y.)
|November 21, 2015
PubMed
まとめ

ニューロン内の低酸素誘導因子1 (HIF-1) の安定化により,腸内信号活性化酵素FMO-2により,ネマトドの寿命が延長されます. セロトニンを含むこの経路は 食事制限に収束し 寿命の維持メカニズムを示唆しています

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