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Magnus Nakrem Lyngbakken1, Julia Brox Skranes1, James A de Lemos1

  • 1From Division of Medicine, Akershus University Hospital, Lørenskog, Norway (M.N.L., J.B.S., H.R., T.O.); Center for Heart Failure Research, Institute of Clinical Medicine, University of Oslo, Oslo, Norway (M.N.L., H.R., T.O.); Division of Cardiology, University of Texas Southwestern Medical Center, Dallas (J.A.d.L.); Bioinformatics Core Facility, Institute for Medical Informatics, Oslo University Hospital and University of Oslo, Oslo, Norway (S.N.); Department of Cardiology, St. Olavs Hospital, Trondheim University Hospital, Trondheim, Norway, and Department of Medicine, Levanger Hospital, Nord-Trøndelag Hospital Trust, Levanger, Norway (H.D.); K.G. Jebsen Center for Exercise in Medicine, Department of Circulation and Medical Imaging, Norwegian University of Science and Technology, Trondheim, Norway (H.D.); and HUNT Research Centre, Department of Public Health and General Practice, Norwegian University of Science and Technology, Levanger, Norway (K.H.).

Circulation
|November 6, 2016
PubMed
まとめ
この要約は機械生成です。

現在の喫煙は心臓のトロポニンI (cTnI) 値の低下と関連しており,たばこ煙が心臓損傷に影響を与える可能性があることを示しています. cTnIの心血管疾患に対する予後値は,非喫煙者と比較して低下しています.

キーワード:
心血管疾患流行病学について予防と対策リスク評価喫煙トロポニン

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科学分野:

  • 心臓病科
  • 心血管研究
  • バイオマーカー

背景:

  • 循環中の心臓トロポニンI (cTnI) とたばこ喫煙は,心血管疾患の確立された危険因子です.
  • 喫煙状況,cTnIレベル,および心血管のアウトカムの関係については,さらなる調査が必要である.

研究 の 目的:

  • 喫煙状況とCTnIレベルとの関連を検証する.
  • 主要な心血管疾患に対する cTnI の予後値に喫煙が影響するかどうかを判断する.

主な方法:

  • HUNT研究から得られたデータを分析し,喫煙者3824人,喫煙者2341人,喫煙者2550人を対象にしました.
  • 前回の心血管疾患や糖尿病のない参加者のcTnIレベルを測定するために使用された高感度アッセイ.
  • cTnI,喫煙,急性心筋梗塞,心不全,心血管疾患による死亡との関連を評価するための予測観察設計.

主要な成果:

  • cTnIレベルは,混同因子を調整した後でも,決して喫煙していない人や元喫煙者に比べ,著しく低かった.
  • cTnI濃度の上昇と心血管疾患との関連は,全体的なコホートでは有意であったが,現在の喫煙者では弱まった.
  • cTnIの心血管疾患の予測精度は,現在の喫煙者において著しく低下し,Framinghamリスクスコアに対するインクリメンタル値は認められませんでした.

結論:

  • 現在の喫煙はcTnI濃度が低く,タバコの煙成分が心筋細胞の損傷に潜在的影響を及ぼすことを示唆しています.
  • cTnIの心血管疾患の予測力は,現在の喫煙者において低下しており,喫煙による心血管疾患は,亜臨床性心筋損傷とは無関係な経路を伴う可能性があることを示唆している.