細菌代謝物による腸内CX3CR1+細胞のGPR31依存型デンドライト突起

Naoki Morita1,2,3, Eiji Umemoto1,2,3, Setsuko Fujita4

  • 1Department of Microbiology and Immunology, Graduate School of Medicine, Osaka University, Suita, Japan.

Nature
|January 25, 2019
PubMed
まとめ

細菌の代謝物質であるピルビック酸と乳酸は,GPR31経由で免疫細胞のデンドライト突起を誘発する. サルモネラ感染に対する免疫反応と耐性を高めます.

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