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Improved Rodent Model of Myocardial Ischemia and Reperfusion Injury
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補足体と中性粒子の活性化が,心筋不全性心筋損傷の病原性において作用する

M H Crawford1, F L Grover, W P Kolb

  • 1Department of Medicine, University of Texas Health Science Center, San Antonio 78284-7872.

Circulation
|December 1, 1988
PubMed
まとめ

筋心臓発作後の補給物の枯渇は,組織損傷とバビオンの心臓発作のサイズを減少させます. この効果は,ポリモルフォニュクレアルの白血球の浸透が減少しただけではありませんが,他の保護メカニズムを示唆しています.

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