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Acute Coronary Syndrome II: Pathophysiology and Clinical Manifestations01:19

Acute Coronary Syndrome II: Pathophysiology and Clinical Manifestations

52
The pathophysiology of Acute Coronary Syndrome [ACD] involves several key processes:The main underlying cause of ACD is atherosclerosis, a chronic inflammatory disease characterized by the buildup of lipid-laden plaques within the coronary arteries.As the atherosclerotic plaque grows in the coronary artery, it may become unstable due to the formation of a lipid-rich core and a thin fibrous cap. Inflammatory cells within the plaque, such as macrophages, secrete enzymes that degrade the...
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Acute Coronary Syndrome III: Diagnostic Studies01:30

Acute Coronary Syndrome III: Diagnostic Studies

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Diagnosing acute coronary syndrome or ACS begins with a thorough patient history. Notable symptoms include central, crushing chest pain radiating to the left arm, neck, jaw, or back, along with shortness of breath, sweating (diaphoresis), nausea, vomiting, dizziness, and palpitations.It is crucial to note any history of cardiac illnesses and assess risk factors, including age, gender, smoking, hypertension, diabetes, hyperlipidemia, and a sedentary lifestyle.During physical examination, vital...
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Acute Coronary Syndrome I: Introduction01:30

Acute Coronary Syndrome I: Introduction

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Acute Coronary Syndrome (ACS) encompasses a spectrum of heart conditions caused by sudden obstruction of coronary arteries, typically resulting from the rupture of an atherosclerotic plaque and subsequent thrombus (blood clot) formation. This obstruction can lead to partial or complete blockage of blood flow, causing varying degrees of myocardial ischemia or infarction.ACS includes the following clinical entities:Unstable Angina (UA)Non-ST-Elevation Myocardial Infarction (NSTEMI)ST-Elevation...
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Angina II: Classification01:27

Angina II: Classification

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Angina, also known as angina pectoris, is a chest pain resulting from diminished blood flow to the heart muscle and is often a symptom of coronary artery disease. Angina presents several variants with distinctive attributes, etiologies, and therapeutic approaches. The main types of angina include stable, unstable, variant (Prinzmetal's), microvascular, intractable, and silent ischemia.Stable angina is caused by atherosclerosis, which leads to the formation of plaques that narrow the coronary...
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Imaging Studies for Cardiovascular System VI: Calcium -Scoring CT01:25

Imaging Studies for Cardiovascular System VI: Calcium -Scoring CT

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Calcium-Scoring CT ScanA calcium-scoring CT scan, also known as coronary artery calcium (CAC) scan, detects calcium deposits in the coronary arteries. This test assesses the risk of coronary artery disease (CAD), which can lead to cardiovascular events such as angina, heart failure, and sudden cardiac arrest.A calcium-scoring CT scan is generally recommended for individuals at intermediate risk of CAD without symptoms. It includes:Men aged 40-75 and women aged 50-75: Especially those with a...
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Aneurysm II: Clinical Manifestations and Diagnostic Studies01:21

Aneurysm II: Clinical Manifestations and Diagnostic Studies

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Thoracic, aortic arch and abdominal aneurysms are significant vascular conditions that can present with various clinical manifestations and lead to serious complications. Understanding these manifestations and the appropriate diagnostic studies is essential for effective management and treatment.Thoracic Aortic AneurysmsThoracic aortic aneurysms often remain asymptomatic until they reach a size that impinges on adjacent structures. They typically cause deep, diffuse chest pain that radiates to...
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  3. 非原因の脆弱性プラークと,stセグメントの上昇と,stセグメントの上昇のない心筋梗塞の予後:prospectiiサブスタディ
  1. ホーム
  3. 非原因の脆弱性プラークと,stセグメントの上昇と,stセグメントの上昇のない心筋梗塞の予後:prospectiiサブスタディ

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A Research Method For Detecting Transient Myocardial Ischemia In Patients With Suspected Acute Coronary Syndrome Using Continuous ST-segment Analysis
18:11

A Research Method For Detecting Transient Myocardial Ischemia In Patients With Suspected Acute Coronary Syndrome Using Continuous ST-segment Analysis

Published on: December 28, 2012

24.4K

非原因の脆弱性プラークと,STセグメントの上昇と,STセグメントの上昇のない心筋梗塞の予後:PROSPECTIIサブスタディ

Pernille G Thrane1,2, Michael Maeng1,2, Akiko Maehara3,4

  • 1Department of Cardiology, Aarhus University Hospital, Denmark (P.G.T., M. Maeng, H.E.B.).

Circulation
|June 23, 2025

PubMed で要約を見る

まとめ
この要約は機械生成です。

高リスクのプラークは,ST上昇心筋梗塞 (STEMI) 患者および非STEMI (NSTEMI) 患者でも同様に見られていました. 未治療の非原発性病変のアウトカムも比較可能で,両方の患者群で同様の再血管化戦略を示唆した.

キーワード:
ST上昇心筋梗塞冠動脈疾患非ST上昇心筋梗塞顕微鏡,近赤外線

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Postconditioning with Lactate-enriched Blood for Cardioprotection in ST-segment Elevation Myocardial Infarction
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科学分野:

  • 心臓病科
  • 介入心臓科
  • 血管生物学

背景:

  • ST上昇性心筋梗塞 (STEMI) と非STEMI (NSTEMI) の非原発性病変に対するリヴァスクラライゼーションについては,臨床ガイドラインが異なる.
  • 高リスクの脆弱性プラークの有病率とSTEMIとNSTEMIにおける結果への影響は十分に理解されていません.

研究 の 目的:

  • STEMI と NSTEMI の患者の非原発性病変における未治療の高リスクの脆弱性プラークの流行を比較する.
  • 2つの患者グループにおけるこれらの非原因の病変に関連した長期的な結果を評価する.

主な方法:

  • PROSPECT II試験には,最近心筋梗塞を患った898人の患者が参加し,3血管血管血管検査を受けた.
  • 近赤外線スペクトル検査と血管内超音波検査により,高リスクプラークの2つの特徴 (プラーク負荷≥70%と脂質核負荷指数≥324.7) が確認されました.
  • 未治療の非原因性病変による主要な心血管疾患 (MACE) は,平均3. 7年間追跡された.

主要な成果:

  • STEMI (12. 8%) とNSTEMI (10. 1%) の2つの特性の高リスクプラークの発生率はわずかに高かったが,患者レベルでは類似していた (38. 8%,32. 7%).
  • STEMIとNSTEMIのグループでは,少なくとも1つの高リスク基準を満たすプラークの有病率も同様でした.
  • STEMI と NSTEMI の患者では,非原因の病変に関連したMACE (8. 6% 対 7. 8%) とすべてのMACE (14. 2% 対 13. 0%) の4年比率が比較された.

    • 結論:

    • STEMIとNSTEMIでは,高リスクの脆弱性プラークの患者あたりの流行率が比較できます.
    • STEMIとNSTEMIの両方において,非原因の損傷から生じる主要な心血管疾患の長期的な発生率は類似しています.
    • これらの発見は,STEMIとNSTEMIの患者における非原因の病変に対する同様の再血管化戦略を支持する.