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Updated: Jun 18, 2026

Visualizing Impairment of the Endothelial and Glial Barriers of the Neurovascular Unit during Experimental Autoimmune Encephalomyelitis In Vivo
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IgA血管炎の子供におけるEndocan,内皮酸化窒素合成体,および溶性CD89の評価

Fehim Akgüngör1, Fatih Temiz1, Yaşar Kandur2

  • 1Department of Pediatrics, School of Medicine, Sutcu Imam University, Kahramanmaras, Türkiye.

Clinical rheumatology
|August 23, 2025
PubMed
まとめ

この研究では,健康な対照群と比較して,Endocan,内皮酸化窒素合成酵素 (eNOS),および溶解性CD89 (sCD89) のレベルが低いことが判明しました. これらの発見は,IGAVにおける内皮損傷のユニークなメカニズムを示唆しています.

キーワード:
エンドカン内皮酸化窒素合成酵素 (eNOS)IgA血管炎溶けるCD89

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