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Updated: May 13, 2026

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P2X7-CaMKIIは5-LOX核転移を誘導し,小下出血後に微小の機能を損なう

Peng-Fei Ding1,2,3, Jia-Tong Zhang2,3, Xiao-Long Zhu2,3

  • 1Department of Neurosurgery, Nanjing Drum Tower Hospital Clinical College of Nanjing Medical University, 321 Zhongshan Road, Nanjing, PR China.

Journal of neuroinflammation
|August 28, 2025
PubMed
まとめ

No abstract available in PubMed .

キーワード:
5-ロックスCa2+ についてLTB4/LXA4比率についてP2X7-CaMKIIサバラクノイド出血

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Antiplatelet drugs emerge as frontline defenders against the insidious threat of thromboembolic diseases, where abnormal clots obstruct vital blood vessels. These drugs stand as bulwarks, inhibiting platelet aggregation and clot formation, thereby mitigating the risk of life-threatening conditions like myocardial infarction, coronary artery disease, and thrombotic strokes.
Prostaglandin synthesis inhibitors, exemplified by the widely known aspirin, wield their power by irreversibly acetylating...