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Updated: May 6, 2026

Studying the Hypothalamic Insulin Signal to Peripheral Glucose Intolerance with a Continuous Drug Infusion System into the Mouse Brain
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子宮内成長制限 (IUGR) は,タンパク質解析によって評価された,成年マウスの全身グルコース負荷に対する下垂体反応を変化させる

Thais A J Mutran1, Amanda P Pedroso1, Adriana P de Souza1

  • 1Department of Physiology, Universidade Federal de São Paulo, São Paulo, Brazil.

Metabolic brain disease
|August 29, 2025
PubMed
まとめ

子宮内成長抑制 (IUGR) は,肥満が発症する前に,下垂体代謝に影響します. 低体内の早期の代謝の変化は,エネルギーホメオスタシスに長期的な悪影響を及ぼす可能性があります.

キーワード:
エネルギーホメオスタシス胎児プログラム下垂体メタボリズム肥満

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Hormones Regulating Blood Glucose01:16

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Insulin is released by beta cells of the pancreas when blood glucose levels are high. It facilitates glucose absorption and utilization in insulin-dependent cells with insulin receptors on their plasma membranes. Insulin promotes glucose uptake by increasing the number of glucose transport proteins in the cell membrane, allowing glucose to enter the cell. As a result, glucose utilization and ATP production are enhanced.
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Carbohydrates consumed through foods are converted into glucose, a crucial energy source for the body. In the prandial state, high blood glucose levels stimulate the secretion of insulin from the pancreas. Insulin inhibits hepatic glucose production and stimulates glucose uptake and metabolism by muscle and adipose tissue. The excess glucose is converted into glycogen and stored in the liver and muscles.
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