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Updated: Jun 14, 2026

Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats
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Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats

Published on: November 20, 2015

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PPROM後の母体と胎児の胎盤の炎症と,その後の新生児の結血症との関連

Janine Zöllkau1, Jana Pastuschek1, Ekkehard Schleußner1

  • 1Department of Obstestrics, Jena University Hospital, Jena, Germany; Center for Sepsis Control and Care (CSCC), Jena University Hospital, Jena, Germany.

Placenta
|August 31, 2025
PubMed
まとめ

No abstract available in PubMed .

キーワード:
EONS についてヒストロジーPPROM について胎盤胎盤の炎症

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Human Placental and Decidual Organ Cultures to Study Infections at the Maternal-fetal Interface07:04

Human Placental and Decidual Organ Cultures to Study Infections at the Maternal-fetal Interface

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A Murine Model of Fetal Exposure to Maternal Inflammation to Study the Effects of Acute Chorioamnionitis on Newborn Intestinal Development08:50

A Murine Model of Fetal Exposure to Maternal Inflammation to Study the Effects of Acute Chorioamnionitis on Newborn Intestinal Development

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関連する概念動画

Teratogenicity01:07

Teratogenicity

The ability of a drug to produce structural deformations and functional abnormalities in the developing embryo or the fetus is called teratogenicity, and the drug producing this effect is known as a teratogen. Teratogenic effects include stillbirth, miscarriage, intrauterine growth restriction, and neurocognitive delay. A teratogen may affect the embryo at different stages of development, which is important in determining the type and extent of the damage. During blastocyst formation, the early...