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Updated: Sep 9, 2025

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S100A9は,DRGニューロンのGPR153/Kcnk16機能を調節することによって,神経疾患の痛みに作用する

Molei Liu1, Dawei Han1, Mingwei Sheng2

  • 1Department of School of Medicine, Nankai University, Tianjin 300071, China.; Department of College of Life Sciences, Nankai University, Tianjin 300071, China.

Cellular signalling
|August 31, 2025
PubMed
まとめ

脊髄細胞の分子であるS100A9は 脊髄根の神経細胞に影響を与えることで 神経病的な痛みを引き起こします S100A9の減少は痛みと炎症を軽減し,治療目標としての可能性を強調します.

キーワード:
背中根のギャングリオンKcnk16 について神経疾患の痛みS100A9,GPR153について

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