m6Aと心臓の転写後の調節:心臓の発達と疾患における新たな役割

Donna Li1, Deqiang Li2,3, Jihyun Jang4,5,6

  • 1Center for Cardiovascular Research, Abigail Wexner Research Institute, Nationwide Children's Hospital, Columbus, OH, USA.

PubMed
まとめ

N6-メチラデノシン (m6A) のような心臓の表写体要因は,心臓の発達と疾患に不可欠です. このレビューでは,心臓のmRNAにおけるそれらの規制的な役割が強調され,将来の研究のための知識のギャップを解決しています.

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