MYB,

Yu Wang1, Chen Li1

  • 1Molecular Testing Center, The First Affiliated Hospital of Jinzhou Medical University, Jinzhou, China.

まとめ

子宮頸がんにおけるMYB過剰発現は,ヘッジホッグシグナル伝達経路を活性化することによって,自然キラー (NK) 細胞の活動を抑制する. この発見は,MYBが子宮頸がんの免疫療法の潜在的な標的であることを示唆しています.

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