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Updated: Sep 9, 2025

Live Cell Calcium Imaging Combined with siRNA Mediated Gene Silencing Identifies Ca2+ Leak Channels in the ER Membrane and their Regulatory Mechanisms
13:40

Live Cell Calcium Imaging Combined with siRNA Mediated Gene Silencing Identifies Ca2+ Leak Channels in the ER Membrane and their Regulatory Mechanisms

Published on: July 7, 2011

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CALB1の静止は,カルシウム媒介のミトコンドリア機能障害と細胞老化を介して前立腺がんの放射線感受性を高めます

Chen Gong1, Senmao Li1, Ye An2

  • 1Department of Urology, The Second Affiliated Hospital of Kunming Medical University, No. 347 Dianmian Street, Wuhua District, Kunming, 650101, Yunnan, China; Urological disease clinical medical center of Yunnan province, The Second Affiliated Hospital of Kunming Medical University, No. 347 Dianmian Street, Wuhua District, Kunming, 650101, Yunnan, China; Scientific and Technological Innovation Team of Basic and Clinical Research of Bladder Cancer in Yunnan Universities, The Second Affiliated Hospital of Kunming Medical University, No. 347 Dianmian Street, Wuhua District, Kunming, 650101, Yunnan, China.

Cell calcium
|September 3, 2025
PubMed
まとめ

カルビンジン1 (CALB1) は前立腺がんの成長と放射線抵抗を誘発する. CALB1の減少は老化を誘発し,放射線に対する感受性を高め,前立腺がんの潜在的な治療標的となる.

キーワード:
CALB1 についてカルシウムホメオスタシス細胞の老化前立腺がん放射線感受性miR-186-5pについて

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