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Updated: May 9, 2026

Myelin Oligodendrocyte Glycoprotein MOG35-55 Induced Experimental Autoimmune Encephalomyelitis EAE in C57BL/6 Mice
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Myelin Oligodendrocyte Glycoprotein MOG35-55 Induced Experimental Autoimmune Encephalomyelitis EAE in C57BL/6 Mice

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ミエリン・オリゴデンドロサイト・グリコプロテイン (MOG) 関連抗体疾患の初期表現としての水頭症:腫瘍を模倣するデミエリネーションの症例報告とレビュー

Makoto Ishii1, Satoko Arai1, Ryotaro Ikeguchi1

  • 1Department of Neurology, Tokyo Women's Medical University, Tokyo, JPN.

Cureus
|September 4, 2025
PubMed
まとめ

ミエリン・オリゴデンドロサイト・グリコプロテイン (MOG) 抗体関連疾患 (MOGAD) は脳腫瘍を模倣し,水頭症を引き起こす可能性があります. 病理学とMOG抗体検査による早期診断は効果的な治療に不可欠です.

キーワード:
脳バイオプシー脳組織病理学脳腫瘍深い灰色の物質脱ミエリン症ゲルミノーマ水頭症ミエリン・オリゴデンドロサイト・抗体関連疾患第 3 室内静止術腫瘍性損傷

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Encephalitis is inflammation of the brain parenchyma, most often due to infections or autoimmune processes. It presents with neuropsychiatric features such as fever, altered mental status, behavioral changes, cognitive dysfunction, seizures, focal deficits, and sometimes autonomic instability. In some cases, the meninges are also involved, resulting in meningoencephalitis.Infectious CausesInfectious encephalitis is most commonly viral but can also result from bacterial, fungal, or parasitic...
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Encephalitis ll: Pathophysiology

Encephalitis is inflammation of the brain parenchyma caused by direct viral invasion or immune-mediated mechanisms triggered by infections or tumors. Both processes lead to neuronal injury, disrupted neurotransmission, and diverse neurological symptoms, often with overlapping clinical and pathological features.Autoimmune EncephalitisIn autoimmune encephalitis, antibodies target neuronal antigens on cell surfaces, synapses, or within neurons. A key example is anti-NMDAR encephalitis, which can...
Cerebral Edema ll: Pathophysiology01:22

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Vasogenic edema is a major form of cerebral edema characterized by abnormal accumulation of fluid in the brain’s extracellular space due to disruption of the blood–brain barrier (BBB). The BBB is a specialized structure composed of endothelial cells connected by tight junctions, supported by astrocytic endfeet and a basement membrane. Under normal conditions, it tightly regulates the movement of ions, proteins, and solutes between the bloodstream and brain parenchyma. When this barrier loses...