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Decreased pulse rate01:14

Decreased pulse rate

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Bradycardia is a medical condition in which the heart rate is slower than normal. It occurs when the heart's natural pacemaker, the sinus node, generates slower electrical impulses than the standard rhythm. In adults, bradycardia is diagnosed when the pulse rate falls below 60 beats per minute, indicating a deviation from the normal heart rate range.
There are specific risk factors that can elevate the likelihood of developing bradycardia. Advanced age is a significant factor, with...
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Dysrhythmias IV: Characteristics of Bradyarrhythmias01:18

Dysrhythmias IV: Characteristics of Bradyarrhythmias

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Bradyarrhythmias are cardiac rhythm disorders characterized by a slower-than-normal heart rate, typically defined as fewer than 60 beats per minute. Some of which are discussed here:Sinus BradycardiaSinus bradycardia presents a heart rate lower than 60 beats per minute, with a regular rhythm originating from the SA node. The ECG typically shows normal P waves preceding each QRS complex, a normal PR interval (0.12 to 0.20 seconds), and a normal QRS duration (0.06 to 0.10 seconds).First-Degree AV...
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Dysrhythmias II: Classification of Tachyarrhythmias01:28

Dysrhythmias II: Classification of Tachyarrhythmias

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Tachyarrhythmias are a type of dysrhythmia where the heart rate exceeds 100 beats per minute. Here are some common types of tachyarrhythmias:Sinus TachycardiaSinus tachycardia originates from increased impulses from the sinus node, leading to an elevated heart rate. It is often triggered by stress, fever, or exercise.Patients may experience palpitations, a sensation of a racing heart, dizziness, and chest discomfort.Causes and Risk Factors: Common causes include physical exertion, emotional...
471
Mechanism of Cardiac Arrhythmias01:28

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Arrhythmias are irregular heart rhythms occurring when the heart's electrical impulses become abnormal. These disturbances can lead to various symptoms, depending on their severity and the underlying cause. Some common factors contributing to arrhythmias include hypoxia, ischemia, electrolyte imbalances, excessive catecholamine exposure, drug toxicity, and muscle overstretching. Arrhythmias can be classified into two main types based on the rate and site of origin of abnormal heart rhythms.
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Disturbances in Heart Rhythm01:29

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Arrhythmia or dysrhythmia refers to an abnormal heart rhythm caused by a defect in the heart's conduction system. It can cause the heart to beat irregularly, too quickly, or too slowly, leading to symptoms like chest pain, shortness of breath, and fainting. Factors such as stress, caffeine, alcohol, nicotine, cocaine, certain drugs, congenital defects, diseases, and electrolyte abnormalities can trigger arrhythmias.
Arrhythmias are categorized by their speed, rhythm, and origin. A slow heart...
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Dysrhythmias VI: Management of Dysrhythmias01:25

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Dysrhythmia management involves a multifaceted approach, incorporating pharmacological treatments, medical procedures, surgical interventions, lifestyle modifications, and patient education.Pharmacological ManagementAntiarrhythmic Drugs:Class I (Sodium Channel Blockers): This class includes quinidine and procainamide, which reduce the speed of impulse conduction in the heart, stabilize the cardiac membrane, and control arrhythmias. Quinidine and procainamide are Class IA agents that prolong the...
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スポーツ 選手 の ブラディカルディア: 罹患 率,メカニズム,リスク

Paolo D'Ambrosio1,2,3, Jarne De Paepe4,5,6,7, Luke W Spencer1,2

  • 1Department of Medicine, The University of Melbourne, Parkville, VIC, Australia (P.D., L.W.S., A.M.M., M.D.F., S.J.R., P.M.K., J.M.K., A.L.G.).

Circulation
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PubMed
まとめ

耐久力スポーツ選手は 運動能力や遺伝子のせいで 心拍が遅いシヌスブラディカルディアを 患うことが多いのです 心拍数が低いポリジェニックリスクスコア (HR-PRS) は,運動選手の心拍数が低いことと関連しており,遺伝子が運動能力に影響を及ぼすことを示唆しています.

キーワード:
AVブロックスポーツ選手ブラディカルディア遺伝学休止する

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科学分野:

  • 心臓病科
  • スポーツ医学
  • 遺伝学

背景:

  • シヌスブラディカルディアは 耐久力アスリートに共通して生理学的適応に起因する.
  • アスリート性ブラディカルディアの遺伝的根拠は ほとんど未知のままです

研究 の 目的:

  • エリートな耐久力アスリートにおける ブラディカルディアの遺伝的寄与を調べるため
  • アスリートにおける心拍数ポリジェニックリスクスコア (HR-PRS) とブラジカルディアの関連性を評価する.

主な方法:

  • 心臓画像,運動テスト,ホルターモニタリングを用いて 465人のエリート耐久力アスリートをフェノタイプした.
  • 有効化されたHR-PRSを用いて遺伝的感受性を評価し,アスリートと非アスリートを比較した.
  • ブラディカード運動選手と非ブラディカード運動選手を比較した.

主要な成果:

  • 運動選手の38%は最低心拍数 ≤40bpmを示し,2%はHR ≤30bpmを示した.
  • HR-PRSの低下は,最低心拍数低下と静止ブラディカルディアの増加と有意に関連していた (OR, 2. 2).
  • ブラディカルディアと休止はよく耐えられ,5. 5年以上の有害な結果と関連しませんでした.

結論:

  • フィットネスと遺伝的変異は 耐久性アスリートのシヌスノード機能に寄与する.
  • HR-PRSは,アスリートと非アスリート間で異なっており,アスリート性の潜在的な遺伝的決定要因を示唆しています.