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Hong-Yan Qian1,2,3, Yan He1,2,3, Chao-Qiong Deng1

  • 1Department of Rheumatology and Clinical Immunology, the First Affiliated Hospital of Xiamen University, School of Medicine, Xiamen University, Xiamen, 361000, China.

Acta pharmacologica Sinica
|January 1, 2026
PubMed
まとめ

No abstract available in PubMed .

キーワード:
FAPαフィブロブラストメソトレクサートリウマチ性関節炎標的型療法

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Tumor Necrosis Factor (TNF), a proinflammatory cytokine, contributes significantly to the inflammation seen in Crohn's disease. It exists as soluble TNF and membrane-bound TNF, with actions mediated through TNF receptors (TNFR). TNFR activation leads to the release of proinflammatory cytokines, T-cell activation, collagen production, and leukocyte migration, all contributing to inflammation in Crohn's disease. Anti-TNF monoclonal antibodies, namely infliximab (Remicade), adalimumab...
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