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Updated: Jan 7, 2026

Studying TGF-β Signaling and TGF-β-induced Epithelial-to-mesenchymal Transition in Breast Cancer and Normal Cells
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三重陰性乳がんにおけるMIR503HG制御遺伝子に対するグルコースの分子効果

Victoria A Reid1, Barbara Yang1,2, Kyle Russo3

  • 1Center of Emphasis in Cancer, Department of Molecular and Translational Medicine, Paul L. Foster School of Medicine, Texas Tech University Health Sciences Center El Paso, El Paso, TX, United States.

British journal of biomedical science
|January 2, 2026
PubMed
まとめ

No abstract available in PubMed .

キーワード:
RNA-seq について遺伝子発現グルコースlncRNA についてトリプルネガティブ乳がん

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Carbohydrates consumed through foods are converted into glucose, a crucial energy source for the body. In the prandial state, high blood glucose levels stimulate the secretion of insulin from the pancreas. Insulin inhibits hepatic glucose production and stimulates glucose uptake and metabolism by muscle and adipose tissue. The excess glucose is converted into glycogen and stored in the liver and muscles.
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