BML-111は,ACE2を活性化することによって,ラットにおけるフォスゲン誘発の急性肺損傷を軽減する

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Updated: Jan 7, 2026

A Model of Self-limited Acute Lung Injury by Unilateral Intra-bronchial Acid Instillation
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A Model of Self-limited Acute Lung Injury by Unilateral Intra-bronchial Acid Instillation

Published on: August 30, 2019

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Xiaojie Guo1, Zi Long2, Minjie Shi1

  • 1Department of Toxicology, Shaanxi Provincial Key Lab of Free Radical Biology and Medicine, Ministry of Education Key Lab of Hazard Assessment and Control in Special Operational Environment, School of Public Health, Fourth Military Medical University, Xi'an, China.

Inhalation toxicology
|January 2, 2026
PubMed
まとめ

No abstract available in PubMed .

キーワード:
ACE2 についてALI についてBML-111 についてフォスゲン

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Anticholinesterases, also known as cholinesterase inhibitors, work by blocking the breakdown of acetylcholine, leading to its accumulation in the synaptic cleft. This accumulation indirectly enhances both muscarinic and nicotinic actions. These agents are classified as reversible or irreversible based on their mechanism of action.     
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